Paper41660629
Differential composition of lymphocyte subpopulations and activation between the hypertensive Bph/2 and normotensive Bpn/3 mouse strains.
Dattmore D and 8 others. Front Immunol, 2025. Cited by 0 papers, by iCite.
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ConductScience Library. Differential composition of lymphocyte subpopulations and activation between the hypertensive Bph/2 and normotensive Bpn/3 mouse strains. Papers, record 41660629. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41660629
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Lymphocyte Subsets | Major topic |
| Hypertension | Major topic |
| Lymphocyte Activation | Major topic |
| Animals | |
| Mice | |
| Cells, Cultured | |
| Blood Pressure | |
| Body Weight | |
| CD4-CD8 Ratio | |
| Immunoglobulin D | |
| Spleen | |
| Cytokines | |
| Cell Proliferation |
Source PubMed
Grants1
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| P01 HL152951 | NHLBI NIH HHS | United States | P01HL152951 | PubMed |
Source NIH RePORTER
References38
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 38,706,961 | Hypertension Increases Susceptibility to Experimental Malaria in Mice. | Function (Oxf) | 2024 | https://conductscience.org/library/papers/38706961 | PubMed |
| 2 | 30,581,433 | Transcriptional and Epigenetic Regulation of Effector and Memory CD8 T Cell Differentiation. | Front Immunol | 2018 | https://conductscience.org/library/papers/30581433 | PubMed |
| 3 | 9,241,632 | Characterization of hypertensive and hypotensive inbred strains of mice. | Lab Anim Sci | 1997 | https://conductscience.org/library/papers/9241632 | PubMed |
| 4 | 10,429,672 | B cell development in the spleen takes place in discrete steps and is determined by the quality of B cell receptor-derived signals. | J Exp Med | 1999 | https://conductscience.org/library/papers/10429672 | PubMed |
| 5 | 23,348,416 | Marginal zone B cells: virtues of innate-like antibody-producing lymphocytes. | Nat Rev Immunol | 2013 | https://conductscience.org/library/papers/23348416 | PubMed |
| 6 | 8,238,975 | Immune system of the spontaneously hypertensive rat: II. Morphology and function. | Anat Rec | 1993 | https://conductscience.org/library/papers/8238975 | PubMed |
| 7 | 4,833,575 | Selection for blood pressure levels in mice. | Genetics | 1974 | https://conductscience.org/library/papers/4833575 | PubMed |
| 8 | 18,725,575 | B lymphocytes: how they develop and function. | Blood | 2008 | https://conductscience.org/library/papers/18725575 | PubMed |
| 9 | 17,875,676 | Role of the T cell in the genesis of angiotensin II induced hypertension and vascular dysfunction. | J Exp Med | 2007 | https://conductscience.org/library/papers/17875676 | PubMed |
| 10 | 5,019,542 | Spontaneous hypertension in laboratory animals. A review of the genetic implications. | J Hered | 1972 | https://conductscience.org/library/papers/5019542 | PubMed |
| 11 | 301,090 | Surface immunoglobulins of lipopolysaccharide-stimulated spleen cells. The behavior of IgM, IgD and IgG. | Eur J Immunol | 1977 | https://conductscience.org/library/papers/301090 | PubMed |
| 12 | 5,970,192 | Systolic blood pressure in eight inbred strains of mice. | Nature | 1966 | https://conductscience.org/library/papers/5970192 | PubMed |
| 13 | 1,773,525 | The role of dominance and epistasis in the genetic control of blood pressure in rodent models of hypertension. | Clin Exp Hypertens A | 1991 | https://conductscience.org/library/papers/1773525 | PubMed |
| 14 | 5,860,215 | Heritability of blood pressure in mice. | J Hered | 1965 | https://conductscience.org/library/papers/5860215 | PubMed |
| 15 | 31,681,017 | Mechanisms Responsible for Genetic Hypertension in Schlager BPH/2 Mice. | Front Physiol | 2019 | https://conductscience.org/library/papers/31681017 | PubMed |
| 16 | 1,831,615 | T-cell memory: the connection between function, phenotype and migration pathways. | Immunol Today | 1991 | https://conductscience.org/library/papers/1831615 | PubMed |
| 17 | 25,411,432 | IgH chain class switch recombination: mechanism and regulation. | J Immunol | 2014 | https://conductscience.org/library/papers/25411432 | PubMed |
| 18 | 11,788,432 | Reduction of renal immune cell infiltration results in blood pressure control in genetically hypertensive rats. | Am J Physiol Renal Physiol | 2002 | https://conductscience.org/library/papers/11788432 | PubMed |
| 19 | 1,831,650 | The expression of B cell surface receptors. III. The murine low-affinity IgE Fc receptor is not expressed on Ly 1 or 'Ly 1-like' B cells. | Int Immunol | 1991 | https://conductscience.org/library/papers/1831650 | PubMed |
| 20 | 6,040,652 | Passive transfer of autoimmune induced hypertension in the rat by lymph node cells. | Tex Rep Biol Med | 1967 | https://conductscience.org/library/papers/6040652 | PubMed |
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The date of each source this release read.
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- Trials
- Comments and corrections
- Cited by
- Genes
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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