Paper · 41686830

A VPS33B CRISPR knockout study: In vitro evidence of an adhesion defect.

Caluianu M and 1 other. PLoS One, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. A VPS33B CRISPR knockout study: In vitro evidence of an adhesion defect. Papers, record 41686830. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41686830

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: A VPS33B CRISPR knockout study: In vitro evidence of an adhesion defect.; PubMed
Journal: PLoS One; PubMed
Year: 2026; PubMed
Publication types: Journal Article, Research Support, Non-U.S. Gov't; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/41686830/; PubMed
DOI: https://doi.org/10.1371/journal.pone.0343240; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC12904430/; PubMed
The journal: https://conductscience.org/library/journal-index/101285081; PubMed
Citations, by iCite: 0; iCite
Revised in PubMed: 2026-11-03; PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Caluianu M	Maria		https://orcid.org/0000-0002-0269-9164
2	Owen KA	Kimberley A

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Vesicular Transport Proteins	Major topic
Cell Adhesion	Major topic
Kidney Tubules, Proximal	Major topic
Humans
Cell Line
Gene Knockout Techniques
Animals
Epithelial Cells

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References38

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	35,151,346	Novel gene mutations in three Japanese patients with ARC syndrome associated mild phenotypes: a case series.	J Med Case Rep	2022	https://conductscience.org/library/papers/35151346	PubMed
2	28,082,148	Vps33b is crucial for structural and functional hepatocyte polarity.	J Hepatol	2017	https://conductscience.org/library/papers/28082148	PubMed
3	37,941,138	WikiPathways 2024: next generation pathway database.	Nucleic Acids Res	2024	https://conductscience.org/library/papers/37941138	PubMed
4	31,501,156	Mechanism of platelet α-granule biogenesis: study of cargo transport and the VPS33B-VPS16B complex in a model system.	Blood Adv	2019	https://conductscience.org/library/papers/31501156	PubMed
5	37,941,124	The Reactome Pathway Knowledgebase 2024.	Nucleic Acids Res	2024	https://conductscience.org/library/papers/37941124	PubMed
6	23,023,225	Integrins β1 and β3 exhibit distinct dynamic nanoscale organizations inside focal adhesions.	Nat Cell Biol	2012	https://conductscience.org/library/papers/23023225	PubMed
7	22,753,090	Associations among genotype, clinical phenotype, and intracellular localization of trafficking proteins in ARC syndrome.	Hum Mutat	2012	https://conductscience.org/library/papers/22753090	PubMed
8	20,190,753	Mutations in VIPAR cause an arthrogryposis, renal dysfunction and cholestasis syndrome phenotype with defects in epithelial polarization.	Nat Genet	2010	https://conductscience.org/library/papers/20190753	PubMed
9	23,918,659	Vps33b pathogenic mutations preferentially affect VIPAS39/SPE-39-positive endosomes.	Hum Mol Genet	2013	https://conductscience.org/library/papers/23918659	PubMed
10	29,409,756	VPS33B and VIPAR are essential for epidermal lamellar body biogenesis and function.	Biochim Biophys Acta Mol Basis Dis	2018	https://conductscience.org/library/papers/29409756	PubMed

1 to 10 of 38

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

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ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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