Paper · 4241817
Effect of hypothyroidism on ionic metabolism and Na-K activated ATP phosphohydrolase activity in the developing rat brain.
Valcana T and 1 other. J Neurochem, 1969. Cited by 83 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Effect of hypothyroidism on ionic metabolism and Na-K activated ATP phosphohydrolase activity in the developing rat brain. Papers, record 4241817. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/4241817
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Effect of hypothyroidism on ionic metabolism and Na-K activated ATP phosphohydrolase activity in the developing rat brain.
- PubMed
Topics18
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Adenosine Triphosphatases | Major topic |
| Cerebellum | Major topic |
| Cerebral Cortex | Major topic |
| Hypothyroidism | Major topic |
| Water-Electrolyte Balance | Major topic |
| Animals | |
| Animals, Newborn | |
| Brain | |
| Calcium | |
| Centrifugation | |
| Chlorides | |
| Electrolytes | |
| Magnesium | |
| Microsomes | |
| Potassium | |
| Rats | |
| Sodium | |
| Thyroxine |
Source PubMed
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Trials0
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Comments and corrections0
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References0
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Cited by8
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 83 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 35,456,949 | Deficiency of Thyroid Hormone Reduces Voltage-Gated Na+ Currents as Well as Expression of Na+/K+-ATPase in the Mouse Hippocampus. | Int J Mol Sci | 2022 | https://conductscience.org/library/papers/35456949 | PubMed |
| 31,010,095 | Fluoride Exposure Induces Inhibition of Sodium-and Potassium-Activated Adenosine Triphosphatase (Na+, K+-ATPase) Enzyme Activity: Molecular Mechanisms and Implications for Public Health. | Int J Environ Res Public Health | 2019 | https://conductscience.org/library/papers/31010095 | PubMed |
| 25,018,677 | Brain Na(+), K(+)-ATPase Activity In Aging and Disease. | Int J Biomed Sci | 2014 | https://conductscience.org/library/papers/25018677 | PubMed |
| 8,139,768 | Subcellular distribution of carbonic anhydrase and Na+,K(+)-ATPase in the brain of the hyt/hyt hypothyroid mice. | Neurochem Res | 1994 | https://conductscience.org/library/papers/8139768 | PubMed |
| 5,289,002 | Mechanism of thyroid calorigenesis: role of active sodium transport. | Proc Natl Acad Sci U S A | 1970 | https://conductscience.org/library/papers/5289002 | PubMed |
| 4,673,705 | Development of potassium effects on ion concentrations and indicator spaces in rat brain-cortex slices during postnatal ontogenesis. | Exp Brain Res | 1972 | https://conductscience.org/library/papers/4673705 | PubMed |
| 4,260,122 | Thyroxine-induced stimulation of hepatic cell transport of calcium and magnesium. | J Clin Invest | 1972 | https://conductscience.org/library/papers/4260122 | PubMed |
| 4,252,666 | The mechanism of the calorigenic action of thyroid hormone. Stimulation of Na plus + K plus-activated adenosinetriphosphatase activity. | J Gen Physiol | 1971 | https://conductscience.org/library/papers/4252666 | PubMed |
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None in this release.
Drugs0
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None in this release.
Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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