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Paper · 42439637

Unveiling the Mysteries of CLEC3B: Physiological Roles, Pathological Impacts, and Research Gaps.

Li L and 1 other. Cells, 2026. Cited by 1 paper, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Unveiling the Mysteries of CLEC3B: Physiological Roles, Pathological Impacts, and Research Gaps. Papers, record 42439637. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42439637

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Unveiling the Mysteries of CLEC3B: Physiological Roles, Pathological Impacts, and Research Gaps.
PubMed
Journal
Cells
PubMed
Year
2026
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
1
iCite
Revised in PubMed
2026-08-13
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Li LLe
2Guo LLianghttps://orcid.org/0000-0001-7353-8336

Source PubMed

Topics4

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Lectins, C-TypeMajor topic
Humans
Animals
Neoplasms

Source PubMed

Grants5

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated by
82571021National Natural Science Foundation of ChinaPubMed
BJJY2025018Shanghai Oriental Talents ProgramPubMed
25ZR1401328Shanghai Natural Science FoundationPubMed
TP2022100Program for Overseas High-level talents at Shanghai Institutions of Higher LearningPubMed
2025KF001Key Laboratory of Exercise and Health Sciences (Shanghai University of Sport), Ministry of EducationPubMed

Source PubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References125

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
133,134,320Comprehensive Analysis of the Relationships Between Tumor Mutation Burden With Immune Infiltrates in Cervical Cell Carcinoma.Front Mol Biosci2020https://conductscience.org/library/papers/33134320PubMed
28,929,539Loss of fibrinogen rescues mice from the pleiotropic effects of plasminogen deficiency.Cell1996https://conductscience.org/library/papers/8929539PubMed
32,506,666Calcium-dependent binding of tetranectin to fibrin.Thromb Res1989https://conductscience.org/library/papers/2506666PubMed
441,526,921
Decoding the tumor microenvironment remodeling orchestrated by CLEC3B+ inflammatory cancer-associated fibroblasts in lung adenocarcinoma immunotherapy: elucidation from pan-cancer spatially single-cell transcriptomics landscape.
J Transl Med2026https://conductscience.org/library/papers/41526921PubMed
540,487,169Integrating gene demethylation and immune modulation: PD-1 nanovesicles as a dual-action therapy for NSCLC.Mater Today Bio2025https://conductscience.org/library/papers/40487169PubMed
638,487,549Serum myostatin as a candidate disease severity and progression biomarker of spinal muscular atrophy.Brain Commun2024https://conductscience.org/library/papers/38487549PubMed
731,477,130Downregulation of exosomal CLEC3B in hepatocellular carcinoma promotes metastasis and angiogenesis via AMPK and VEGF signals.Cell Commun Signal2019https://conductscience.org/library/papers/31477130PubMed
830,894,065Cancer-associated fibroblasts promote colorectal cancer progression by secreting CLEC3B.Cancer Biol Ther2019https://conductscience.org/library/papers/30894065PubMed
935,500,520Down-regulation of CLEC3B facilitates epithelial-mesenchymal transition, migration and invasion of lung adenocarcinoma cells.Tissue Cell2022https://conductscience.org/library/papers/35500520PubMed
107,542,656Diversity of function is inherent in matricellular proteins: an appraisal of thrombospondin 1.J Cell Biol1995https://conductscience.org/library/papers/7542656PubMed

1 to 10 of 125

See all 125 references

Cited by1

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 1 citation.

PMIDTitleJournalYearIn Papers
42,589,387Tetranectin: Molecular Mechanisms, Biological Functions and Clinical Implications.Int J Mol Sci2026https://conductscience.org/library/papers/42589387

Source PubMed

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
CLEC3B7,123PubTator's text mininghttps://conductscience.org/library/genes/7123

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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