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Paper · 42542977

Mechanisms and Manifestations of Ligand Bias in Signaling: Focus on Receptor Tyrosine Kinases.

Schuck RJ and 4 others. Chem Rev, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Mechanisms and Manifestations of Ligand Bias in Signaling: Focus on Receptor Tyrosine Kinases. Papers, record 42542977. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42542977

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Mechanisms and Manifestations of Ligand Bias in Signaling: Focus on Receptor Tyrosine Kinases.
PubMed
Journal
Chem Rev
PubMed
Year
2026
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
0
iCite
Revised in PubMed
2026-08-16
PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Schuck RJRyan J
2Roy TTanaya
3Bondar ANAna-Nicoletahttps://orcid.org/0000-0003-2636-9773
4Krejci PPavel
5Hristova KKalinahttps://orcid.org/0000-0003-4274-4406

Source PubMed

Topics5

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Signal TransductionMajor topic
Receptor Protein-Tyrosine KinasesMajor topic
Ligands
Humans
Animals

Source PubMed

Grants12

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
NAAkademie Ved Cesk?? RepublikyPubMedPubMed
NAMinisterstvo ??kolstv??, Ml??de??e a Telov??chovyPubMedPubMed
PNRR-III-C9-2022-I5-19Ministerul Cercetarii, Inovarii si DigitalizariiPubMedPubMed
NAAgentura Pro Zdravotnick?? V??zkum Cesk?? RepublikyPubMedPubMed
NU23-10-00550Agentura Pro Zdravotnick?? V??zkum Cesk?? RepublikyPubMedPubMed
2451634National Science FoundationPubMedPubMed
101086667European CommissionPubMedPubMed
GM 131374U.S. Department of Health and Human ServicesPubMedPubMed
R01 GM131374NIGMS NIH HHSUnited StatesR01GM131374PubMedNIH RePORTER
LUAUS23295Ministerstvo ??kolstv??, Ml??de??e a Telov??chovyPubMedPubMed

1 to 10 of 12

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Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References322

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
127,052,508VEGFR-2 conformational switch in response to ligand binding.Elife2016https://conductscience.org/library/papers/27052508PubMed
216,597,617Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family.J Biol Chem2006https://conductscience.org/library/papers/16597617PubMed
337,243,489Targeting the EGFR signaling pathway in cancer therapy: What's new in 2023?Expert Opin Ther Targets2023https://conductscience.org/library/papers/37243489PubMed
427,825,441Neurotrophin Signaling Is Required for Glucose-Induced Insulin Secretion.Dev Cell2016https://conductscience.org/library/papers/27825441PubMed
520,228,801An extracellular steric seeding mechanism for Eph-ephrin signaling platform assembly.Nat Struct Mol Biol2010https://conductscience.org/library/papers/20228801PubMed
612,461,170A putative molecular-activation switch in the transmembrane domain of erbB2.Proc Natl Acad Sci U S A2002https://conductscience.org/library/papers/12461170PubMed
727,083,447BMP2 induces chondrogenic differentiation, osteogenic differentiation and endochondral ossification in stem cells.Cell Tissue Res2016https://conductscience.org/library/papers/27083447PubMed
837,996,538Eph receptors and ephrins in cancer progression.Nat Rev Cancer2024https://conductscience.org/library/papers/37996538PubMed
917,715,395Monitoring interactions between receptor tyrosine kinases and their downstream effector proteins in living cells using bioluminescence resonance energy transfer.Mol Pharmacol2007https://conductscience.org/library/papers/17715395PubMed
1011,668,614
Congenital insensitivity to pain with anhidrosis (CIPA): novel mutations of the TRKA (NTRK1) gene, a putative uniparental disomy, and a linkage of the mutant TRKA and PKLR genes in a family with CIPA and pyruvate kinase deficiency.
Hum Mutat2001https://conductscience.org/library/papers/11668614PubMed

1 to 10 of 322

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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