Paper · 42550307
Routes to colorectal cancer in Lynch syndrome: a decade of molecular and clinical insights converging on Schrödinger's cat.
Ahadova A and 4 others. Fam Cancer, 2026. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Routes to colorectal cancer in Lynch syndrome: a decade of molecular and clinical insights converging on Schrödinger's cat. Papers, record 42550307. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42550307
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Routes to colorectal cancer in Lynch syndrome: a decade of molecular and clinical insights converging on Schrödinger's cat.
- PubMed
Topics5
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Colorectal Neoplasms, Hereditary Nonpolyposis | Major topic |
| Colorectal Neoplasms | Major topic |
| Humans | |
| Microsatellite Instability | |
| DNA Mismatch Repair |
Source PubMed
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References86
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 31,926,173 | Associations of Pathogenic Variants in MLH1, MSH2, and MSH6 With Risk of Colorectal Adenomas and Tumors and With Somatic Mutations in Patients With Lynch Syndrome. | Gastroenterology | 2020 | https://conductscience.org/library/papers/31926173 | PubMed |
| 2 | 33,243,186 | Correction to: The majority of β-catenin mutations in colorectal cancer is homozygous. | BMC Cancer | 2020 | https://conductscience.org/library/papers/33243186 | PubMed |
| 3 | 41,674,481 | A multilevel perspective on MSH6-associated Lynch syndrome: Integrating molecular, biological, and clinical insights. | Int J Cancer | 2026 | https://conductscience.org/library/papers/41674481 | PubMed |
| 4 | 41,193,656 | Epigenetically driven and early immune evasion in colorectal cancer evolution. | Nat Genet | 2025 | https://conductscience.org/library/papers/41193656 | PubMed |
| 5 | 33,310,086 | A High Percentage of Early-age Onset Colorectal Cancer Is Potentially Preventable. | Gastroenterology | 2021 | https://conductscience.org/library/papers/33310086 | PubMed |
| 6 | 36,251,018 | A simple approach for detecting HLA-A*02 alleles in archival formalin-fixed paraffin-embedded tissue samples and an application example for studying cancer immunoediting. | HLA | 2023 | https://conductscience.org/library/papers/36251018 | PubMed |
| 7 | 24,685,131 | The APC network regulates the removal of mutated cells from colonic crypts. | Cell Rep | 2014 | https://conductscience.org/library/papers/24685131 | PubMed |
| 8 | 26,657,901 | Cancer incidence and survival in Lynch syndrome patients receiving colonoscopic and gynaecological surveillance: first report from the prospective Lynch syndrome database. | Gut | 2017 | https://conductscience.org/library/papers/26657901 | PubMed |
| 9 | 41,339,549 | Decay of driver mutations shapes the landscape of intestinal transformation. | Nature | 2026 | https://conductscience.org/library/papers/41339549 | PubMed |
| 10 | 32,534,647 | Cancer prevention with aspirin in hereditary colorectal cancer (Lynch syndrome), 10-year follow-up and registry-based 20-year data in the CAPP2 study: a double-blind, randomised, placebo-controlled trial. | Lancet | 2020 | https://conductscience.org/library/papers/32534647 | PubMed |
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None in this release.
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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