Paper · 42653285
General Assessment of Indole Derivatives and Wnt Pathway as Potential Key Factors Against Various Diseases, with Particular Emphasis on Thyroid Tumors.
Skoczyńska AK and 2 others. Int J Mol Sci, 2026. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
Cite this record
ConductScience Library. General Assessment of Indole Derivatives and Wnt Pathway as Potential Key Factors Against Various Diseases, with Particular Emphasis on Thyroid Tumors. Papers, record 42653285. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42653285
On this page
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- General Assessment of Indole Derivatives and Wnt Pathway as Potential Key Factors Against Various Diseases, with Particular Emphasis on Thyroid Tumors.
- PubMed
Topics6
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Thyroid Neoplasms | Major topic |
| Indoles | Major topic |
| Wnt Signaling Pathway | Major topic |
| Humans | |
| Animals | |
| Antineoplastic Agents |
Source PubMed
Grants1
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| 503/1-107-03/503-11-001 | Medical University of Lodz | PubMed |
Source PubMed
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References179
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 27,174,043 | Molecular genetics of thyroid cancer. | Genet Res (Camb) | 2016 | https://conductscience.org/library/papers/27174043 | PubMed |
| 2 | 16,203,150 | Synthesis of functionalized 2-aryl-5-nitro-1H-indoles and their activity as bacterial NorA efflux pump inhibitors. | Bioorg Med Chem | 2006 | https://conductscience.org/library/papers/16203150 | PubMed |
| 3 | 1,643,248 | Oligomerization of indole-3-carbinol in aqueous acid. | Chem Res Toxicol | 1992 | https://conductscience.org/library/papers/1643248 | PubMed |
| 4 | 17,230,199 | Frizzled/PCP signalling: a conserved mechanism regulating cell polarity and directed motility. | Nat Rev Genet | 2007 | https://conductscience.org/library/papers/17230199 | PubMed |
| 5 | 14,587,035 | Melatonin reduces Fenton reaction-induced lipid peroxidation in porcine thyroid tissue. | J Cell Biochem | 2003 | https://conductscience.org/library/papers/14587035 | PubMed |
| 6 | 35,593,680 | Diagnosis and treatment of thyroid cancer in adult patients - Recommendations of Polish Scientific Societies and the National Oncological Strategy. 2022 Update [Diagnostyka i leczenie raka tarczycy u chorych dorosłych - Rekomendacje Polskich Towarzystw Naukowych oraz Narodowej Strategii Onkologicznej. Aktualizacja na rok 2022]. | Endokrynol Pol | 2022 | https://conductscience.org/library/papers/35593680 | PubMed |
| 7 | 7,592,064 | Sattabacins and sattazolins: new biologically active compounds with antiviral properties extracted from a Bacillus sp. | J Antibiot (Tokyo) | 1995 | https://conductscience.org/library/papers/7592064 | PubMed |
| 8 | 15,573,866 | Sigma receptor ligands: applications in inflammation and oncology. | Curr Opin Investig Drugs | 2004 | https://conductscience.org/library/papers/15573866 | PubMed |
| 9 | 11,975,483 | New antiinfective and human 5-HT2 receptor binding natural and semisynthetic compounds from the Jamaican sponge Smenospongia aurea. | J Nat Prod | 2002 | https://conductscience.org/library/papers/11975483 | PubMed |
| 10 | 29,525,603 | Melatonin suppresses thyroid cancer growth and overcomes radioresistance via inhibition of p65 phosphorylation and induction of ROS. | Redox Biol | 2018 | https://conductscience.org/library/papers/29525603 | PubMed |
1 to 10 of 179
See all 179 referencesCited by0
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.
None in this release.
Genes1
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes |
|---|---|---|---|
| WNT1 | 7,471 | PubTator's text mining | https://conductscience.org/library/genes/7471 |
Source NCBI Gene
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this paper from software.
The record and each of its lists are JSON, twenty rows a page. No key.
GET https://conductscience.org/api/papers/v1/42653285
GET https://conductscience.org/api/papers/v1/42653285/references?page=2