Paper · 42757257
Prenatal stress selectively enhances locomotor hyperactivity in male mice with embryonic loss of Fgfr2.
Chen MX and 3 others. Front Psychiatry, 2026. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Prenatal stress selectively enhances locomotor hyperactivity in male mice with embryonic loss of Fgfr2. Papers, record 42757257. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42757257
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Paper
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- Title
- Prenatal stress selectively enhances locomotor hyperactivity in male mice with embryonic loss of Fgfr2.
- PubMed
Topics0
NLM's MeSH indexing of the paper, its major topics first.
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Grants2
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by | Source |
|---|---|---|---|---|---|
| K08 MH086812 | NIMH NIH HHS | United States | K08MH086812 | PubMed | NIH RePORTER |
| R01 ES035696 | NIEHS NIH HHS | United States | R01ES035696 | PubMed | NIH RePORTER |
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections1
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
| Kind | PMID | Title | In Papers |
|---|---|---|---|
| Update of | 41,676,519 | Prenatal stress interacts with embryonic loss of Fgfr2 to increase locomotor hyperactivity in mice. | https://conductscience.org/library/papers/41676519 |
Source PubMed
References107
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 21,779,823 | Update on environmental risk factors for attention-deficit/hyperactivity disorder. | Curr Psychiatry Rep | 2011 | https://conductscience.org/library/papers/21779823 | PubMed |
| 2 | 19,621,976 | Toward a new understanding of attention-deficit hyperactivity disorder pathophysiology: an important role for prefrontal cortex dysfunction. | CNS Drugs | 2009 | https://conductscience.org/library/papers/19621976 | PubMed |
| 3 | 17,761,882 | A MicroRNA feedback circuit in midbrain dopamine neurons. | Science | 2007 | https://conductscience.org/library/papers/17761882 | PubMed |
| 4 | 7,824,549 | Permanent dopaminergic alterations in the n. accumbens after prenatal stress. | Pharmacol Biochem Behav | 1994 | https://conductscience.org/library/papers/7824549 | PubMed |
| 5 | 36,906,620 | Neonatal loss of FGFR2 in astroglial cells affects locomotion, sociability, working memory, and glia-neuron interactions in mice. | Transl Psychiatry | 2023 | https://conductscience.org/library/papers/36906620 | PubMed |
| 6 | 15,950,004 | Molecular genetics of attention-deficit/hyperactivity disorder. | Biol Psychiatry | 2005 | https://conductscience.org/library/papers/15950004 | PubMed |
| 7 | 33,445,083 | Fibroblast Growth Factor 2 Implicated in Childhood Anxiety and Depression Symptoms. | J Affect Disord | 2021 | https://conductscience.org/library/papers/33445083 | PubMed |
| 8 | 38,778,436 | ADHD Prevalence Among U.S. Children and Adolescents in 2022: Diagnosis, Severity, Co-Occurring Disorders, and Treatment. | J Clin Child Adolesc Psychol | 2024 | https://conductscience.org/library/papers/38778436 | PubMed |
| 9 | 17,355,398 | Antenatal maternal stress and long-term effects on child neurodevelopment: how and why? | J Child Psychol Psychiatry | 2007 | https://conductscience.org/library/papers/17355398 | PubMed |
| 10 | 18,423,592 | The long-term behavioural consequences of prenatal stress. | Neurosci Biobehav Rev | 2008 | https://conductscience.org/library/papers/18423592 | PubMed |
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Genes0
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None in this release.
Drugs0
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None in this release.
Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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