Paper · 42840641

Retinoic acid regulates donor myeloid-derived suppressor cell and colonic dendrite cell reconstitution after allogeneic hematopoietic stem cell transplantation in mice.

Li X and 8 others. Front Immunol, 2026.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Retinoic acid regulates donor myeloid-derived suppressor cell and colonic dendrite cell reconstitution after allogeneic hematopoietic stem cell transplantation in mice. Papers, record 42840641. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42840641

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Retinoic acid regulates donor myeloid-derived suppressor cell and colonic dendrite cell reconstitution after allogeneic hematopoietic stem cell transplantation in mice.
Journal: Front Immunol
Year: 2026
Publication types: Journal Article
PubMed: https://pubmed.ncbi.nlm.nih.gov/42840641/
DOI: https://doi.org/10.3389/fimmu.2026.1599346
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC13640452/
The journal: https://conductscience.org/library/journal-index/101560960
Revised in PubMed: 2026-10-09

Source PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Li X	Xinlei
2	Zheng J	Jianwei
3	Zhou D	Dian
4	Limpert R	Rachel
5	Taylor B	Brian
6	Tian L	Linlu
7	Yu XZ	Xue-Zhong
8	Drobyski WR	William R
9	Chen X	Xiao

Source PubMed

Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Tretinoin	Major topic
Myeloid-Derived Suppressor Cells	Major topic
Dendritic Cells	Major topic
Hematopoietic Stem Cell Transplantation	Major topic
Colon	Major topic
Animals
Mice
Mice, Inbred BALB C
Graft vs Host Disease
Mice, Inbred C57BL
Transplantation, Homologous
Cell Differentiation

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
R01 AI125334	NIAID NIH HHS	United States	R01AI125334	PubMed

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References80

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	30,885,956	Dendritic Cell Expression of Retinal Aldehyde Dehydrogenase-2 Controls Graft-versus-Host Disease Lethality.	J Immunol	2019	https://conductscience.org/library/papers/30885956	PubMed
2	34,465,895	Dendritic cell functions in the inductive and effector sites of intestinal immunity.	Mucosal Immunol	2022	https://conductscience.org/library/papers/34465895	PubMed
3	31,207,088	Radiation and host retinoic acid signaling promote the induction of gut-homing donor T cells after allogeneic hematopoietic stem cell transplantation.	Am J Transplant	2020	https://conductscience.org/library/papers/31207088	PubMed
4	12,930,397	All-trans retinoic acid skews monocyte differentiation into interleukin-12-secreting dendritic-like cells.	Br J Haematol	2003	https://conductscience.org/library/papers/12930397	PubMed
5	17,785,784	In vitro induction of mucosa-type dendritic cells by all-trans retinoic acid.	J Immunol	2007	https://conductscience.org/library/papers/17785784	PubMed
6	24,958,183	Current and emerging strategies for the prevention of graft-versus-host disease.	Nat Rev Clin Oncol	2014	https://conductscience.org/library/papers/24958183	PubMed
7	18,684,916	Retinoic acid increases Foxp3+ regulatory T cells and inhibits development of Th17 cells by enhancing TGF-beta-driven Smad3 signaling and inhibiting IL-6 and IL-23 receptor expression.	J Immunol	2008	https://conductscience.org/library/papers/18684916	PubMed
8	20,807,889	Bone marrow myeloid-derived suppressor cells (MDSCs) inhibit graft-versus-host disease (GVHD) via an arginase-1-dependent mechanism that is up-regulated by interleukin-13.	Blood	2010	https://conductscience.org/library/papers/20807889	PubMed
9	29,171,820	Acute Graft-versus-Host Disease - Biologic Process, Prevention, and Therapy.	N Engl J Med	2017	https://conductscience.org/library/papers/29171820	PubMed
10	32,086,381	Defining the emergence of myeloid-derived suppressor cells in breast cancer using single-cell transcriptomics.	Sci Immunol	2020	https://conductscience.org/library/papers/32086381	PubMed

1 to 10 of 80

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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