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Paper · 42893232

Long-term voluntary exercise partially reverses obesity-associated transcriptomic remodeling in brown adipose tissue.

Sakurai T and 2 others. Metabol Open, 2026.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Long-term voluntary exercise partially reverses obesity-associated transcriptomic remodeling in brown adipose tissue. Papers, record 42893232. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42893232

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Long-term voluntary exercise partially reverses obesity-associated transcriptomic remodeling in brown adipose tissue.
Journal
Metabol Open
Year
2026
Publication types
Journal Article
Revised in PubMed
2026-10-19

Source PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Sakurai TTakuya
2Nozaki EEriko
3Iwawa TTetsuya

Source PubMed

Topics0

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None in this release.

Grants0

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None in this release.

Trials0

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None in this release.

Comments and corrections0

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References29

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
126,526,134TNNI1, TNNI2 and TNNI3: Evolution, regulation, and protein structure-function relationships.Gene2016https://conductscience.org/library/papers/26526134PubMed
217,167,474Inflammation and metabolic disorders.Nature2006https://conductscience.org/library/papers/17167474PubMed
312,499,399Myotilin, the limb-girdle muscular dystrophy 1A (LGMD1A) protein, cross-links actin filaments and controls sarcomere assembly.Hum Mol Genet2003https://conductscience.org/library/papers/12499399PubMed
432,620,768NCLX prevents cell death during adrenergic activation of the brown adipose tissue.Nat Commun2020https://conductscience.org/library/papers/32620768PubMed
514,679,177Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance.J Clin Invest2003https://conductscience.org/library/papers/14679177PubMed
635,977,541The mitochondrial calcium uniporter engages UCP1 to form a thermoporter that promotes thermogenesis.Cell Metab2022https://conductscience.org/library/papers/35977541PubMed
739,745,717Effect of different intensity aerobic exercise on remodeling immune microenvironment of adipose tissue in obesity mouse.Am J Physiol Regul Integr Comp Physiol2025https://conductscience.org/library/papers/39745717PubMed
834,082,784Aerobic exercise promotes the functions of brown adipose tissue in obese mice via a mechanism involving COX2 in the VEGF signaling pathway.Nutr Metab (Lond)2021https://conductscience.org/library/papers/34082784PubMed
919,357,405Cold-activated brown adipose tissue in healthy men.N Engl J Med2009https://conductscience.org/library/papers/19357405PubMed
1033,288,873Calsequestrin: a well-known but curious protein in skeletal muscle.Exp Mol Med2020https://conductscience.org/library/papers/33288873PubMed

1 to 10 of 29

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Cited by0

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Genes0

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None in this release.

Drugs0

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None in this release.

Data0

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None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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