Paper · 42903377
Role of insulin resistance in atherosclerosis: underlying mechanisms and emerging stem cell-based therapeutic strategies.
Zhang Q and 4 others. Front Med (Lausanne), 2026.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Role of insulin resistance in atherosclerosis: underlying mechanisms and emerging stem cell-based therapeutic strategies. Papers, record 42903377. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42903377
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Role of insulin resistance in atherosclerosis: underlying mechanisms and emerging stem cell-based therapeutic strategies.
- Journal
- Front Med (Lausanne)
- Year
- 2026
- Publication types
- Journal Article, Review
- Revised in PubMed
- 2026-10-23
Source PubMed
Topics0
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Grants0
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References133
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 16,581,003 | Macrophage insulin receptor deficiency increases ER stress-induced apoptosis and necrotic core formation in advanced atherosclerotic lesions. | Cell Metab | 2006 | https://conductscience.org/library/papers/16581003 | PubMed |
| 2 | 40,673,552 | Mitochondrial Calcium Uniporter Regulates Metabolic Remodeling and Smooth Muscle Cell Proliferation in Type 2 Diabetes. | J Am Heart Assoc | 2025 | https://conductscience.org/library/papers/40673552 | PubMed |
| 3 | 39,062,066 | The Triglyceride/HDL Ratio as a Surrogate Biomarker for Insulin Resistance. | Biomedicines | 2024 | https://conductscience.org/library/papers/39062066 | PubMed |
| 4 | 15,792,994 | Insulin resistance in spontaneously hypertensive rats is associated with endothelial dysfunction characterized by imbalance between NO and ET-1 production. | Am J Physiol Heart Circ Physiol | 2005 | https://conductscience.org/library/papers/15792994 | PubMed |
| 5 | 15,486,293 | Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes. | Science | 2004 | https://conductscience.org/library/papers/15486293 | PubMed |
| 6 | 30,621,010 | New Therapeutic Implications of Endothelial Nitric Oxide Synthase (eNOS) Function/Dysfunction in Cardiovascular Disease. | Int J Mol Sci | 2019 | https://conductscience.org/library/papers/30621010 | PubMed |
| 7 | 38,553,774 | Persistent epigenetic signals propel a senescence-associated secretory phenotype and trained innate immunity in CD34+ hematopoietic stem cells from diabetic patients. | Cardiovasc Diabetol | 2024 | https://conductscience.org/library/papers/38553774 | PubMed |
| 8 | 39,524,330 | High-fat-diet-induced hepatic insulin resistance per se attenuates murine de novo lipogenesis. | iScience | 2024 | https://conductscience.org/library/papers/39524330 | PubMed |
| 9 | 36,312,002 | Effectiveness and safety of human umbilical cord-mesenchymal stem cells for treating type 2 diabetes mellitus. | World J Diabetes | 2022 | https://conductscience.org/library/papers/36312002 | PubMed |
| 10 | 36,994,866 | Insulin resistance and cardiovascular disease. | J Int Med Res | 2023 | https://conductscience.org/library/papers/36994866 | PubMed |
1 to 10 of 133
See all 133 referencesCited by0
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Genes0
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None in this release.
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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