Paper · 42979207
Liver-Lung Organotropism in Pancreatic Ductal Adenocarcinoma: A Five-Stage Framework.
Cong B and 3 others. Cancer Manag Res, 2026.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Liver-Lung Organotropism in Pancreatic Ductal Adenocarcinoma: A Five-Stage Framework. Papers, record 42979207. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42979207
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Liver-Lung Organotropism in Pancreatic Ductal Adenocarcinoma: A Five-Stage Framework.
- Journal
- Cancer Manag Res
- Year
- 2026
- Publication types
- Journal Article, Review
- Revised in PubMed
- 2026-11-05
Source PubMed
Topics0
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Trials0
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Comments and corrections0
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References67
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 28,092,686 | Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis. | Nat Genet | 2017 | https://conductscience.org/library/papers/28092686 | PubMed |
| 2 | 37,678,671 | Pancreatic cancer: ESMO Clinical Practice Guideline for diagnosis, treatment and follow-up. | Ann Oncol | 2023 | https://conductscience.org/library/papers/37678671 | PubMed |
| 3 | 24,856,585 | Stromal elements act to restrain, rather than support, pancreatic ductal adenocarcinoma. | Cancer Cell | 2014 | https://conductscience.org/library/papers/24856585 | PubMed |
| 4 | 26,560,028 | Epithelial-to-mesenchymal transition is dispensable for metastasis but induces chemoresistance in pancreatic cancer. | Nature | 2015 | https://conductscience.org/library/papers/26560028 | PubMed |
| 5 | 31,483,448 | Patterns of Recurrence After Resection of Pancreatic Ductal Adenocarcinoma: A Secondary Analysis of the ESPAC-4 Randomized Adjuvant Chemotherapy Trial. | JAMA Surg | 2019 | https://conductscience.org/library/papers/31483448 | PubMed |
| 6 | 28,414,315 | The EMT-activator Zeb1 is a key factor for cell plasticity and promotes metastasis in pancreatic cancer. | Nat Cell Biol | 2017 | https://conductscience.org/library/papers/28414315 | PubMed |
| 7 | 35,740,692 | The Liver Pre-Metastatic Niche in Pancreatic Cancer: A Potential Opportunity for Intervention. | Cancers (Basel) | 2022 | https://conductscience.org/library/papers/35740692 | PubMed |
| 8 | 29,675,723 | Intratumoral Heterogeneity of SMAD4 Immunohistochemical Expression and Its Role in Prediction of Recurrence Pattern in Patients with Resectable Pancreatic Cancer. | J Gastrointest Cancer | 2019 | https://conductscience.org/library/papers/29675723 | PubMed |
| 9 | 28,790,031 | BLIMP1 Induces Transient Metastatic Heterogeneity in Pancreatic Cancer. | Cancer Discov | 2017 | https://conductscience.org/library/papers/28790031 | PubMed |
| 10 | 37,526,431 | Isolated pulmonary metastases in pancreatic ductal adenocarcinoma: a review of current evidence. | Cancer Biol Ther | 2023 | https://conductscience.org/library/papers/37526431 | PubMed |
1 to 10 of 67
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Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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