Paper · 42979282
The CD19-Coupled CD21 is an Essential Receptor for B cell Activation.
Zhang G. J Cell Signal, 2026.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. The CD19-Coupled CD21 is an Essential Receptor for B cell Activation. Papers, record 42979282. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42979282
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- The CD19-Coupled CD21 is an Essential Receptor for B cell Activation.
- Journal
- J Cell Signal
- Year
- 2026
- Publication types
- Journal Article
- Revised in PubMed
- 2026-11-05
Source PubMed
Topics0
NLM's MeSH indexing of the paper, its major topics first.
None in this release.
Grants1
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| R01 GM135421 | NIGMS NIH HHS | United States | R01GM135421 | PubMed |
Source NIH RePORTER
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References27
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 1,836,011 | Mapping of the C3b-binding site of CR1 and construction of a (CR1)2-F(ab')2 chimeric complement inhibitor. | J Exp Med | 1991 | https://conductscience.org/library/papers/1836011 | PubMed |
| 2 | 3,088,111 | Synergy between the T3/antigen receptor complex and Tp44 in the activation of human T cells. | J Immunol | 1986 | https://conductscience.org/library/papers/3088111 | PubMed |
| 3 | 4,556,375 | Role of complement in induction of the allergic response. | Nat New Biol | 1972 | https://conductscience.org/library/papers/4556375 | PubMed |
| 4 | 31,461,748 | Structural basis of assembly of the human T cell receptor-CD3 complex. | Nature | 2019 | https://conductscience.org/library/papers/31461748 | PubMed |
| 5 | 1,956,380 | Peripheral deletion of self-reactive B cells. | Nature | 1991 | https://conductscience.org/library/papers/1956380 | PubMed |
| 6 | 35,985,289 | Structure of a fully assembled tumor-specific T cell receptor ligated by pMHC. | Cell | 2022 | https://conductscience.org/library/papers/35985289 | PubMed |
| 7 | 17,709,481 | Cutting edge: Complement (C3d)-linked antigens break B cell anergy. | J Immunol | 2007 | https://conductscience.org/library/papers/17709481 | PubMed |
| 8 | 30,559,744 | B Cell Siglecs-News on Signaling and Its Interplay With Ligand Binding. | Front Immunol | 2018 | https://conductscience.org/library/papers/30559744 | PubMed |
| 9 | 12,486,104 | Non-T cell activation linker (NTAL): a transmembrane adaptor protein involved in immunoreceptor signaling. | J Exp Med | 2002 | https://conductscience.org/library/papers/12486104 | PubMed |
| 10 | 9,480,991 | Inhibition of the B cell by CD22: a requirement for Lyn. | J Exp Med | 1998 | https://conductscience.org/library/papers/9480991 | PubMed |
1 to 10 of 27
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Genes0
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None in this release.
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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