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Paper · 42979282

The CD19-Coupled CD21 is an Essential Receptor for B cell Activation.

Zhang G. J Cell Signal, 2026.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. The CD19-Coupled CD21 is an Essential Receptor for B cell Activation. Papers, record 42979282. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42979282

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
The CD19-Coupled CD21 is an Essential Receptor for B cell Activation.
Journal
J Cell Signal
Year
2026
Publication types
Journal Article
Revised in PubMed
2026-11-05

Source PubMed

Authors1

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Zhang GGongyi

Source PubMed

Topics0

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None in this release.

Grants1

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Grant, as the record writes itAgencyCountryNIH projectStated by
R01 GM135421NIGMS NIH HHSUnited StatesR01GM135421PubMed

Trials0

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None in this release.

Comments and corrections0

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None in this release.

References27

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
11,836,011Mapping of the C3b-binding site of CR1 and construction of a (CR1)2-F(ab')2 chimeric complement inhibitor.J Exp Med1991https://conductscience.org/library/papers/1836011PubMed
23,088,111Synergy between the T3/antigen receptor complex and Tp44 in the activation of human T cells.J Immunol1986https://conductscience.org/library/papers/3088111PubMed
34,556,375Role of complement in induction of the allergic response.Nat New Biol1972https://conductscience.org/library/papers/4556375PubMed
431,461,748Structural basis of assembly of the human T cell receptor-CD3 complex.Nature2019https://conductscience.org/library/papers/31461748PubMed
51,956,380Peripheral deletion of self-reactive B cells.Nature1991https://conductscience.org/library/papers/1956380PubMed
635,985,289Structure of a fully assembled tumor-specific T cell receptor ligated by pMHC.Cell2022https://conductscience.org/library/papers/35985289PubMed
717,709,481Cutting edge: Complement (C3d)-linked antigens break B cell anergy.J Immunol2007https://conductscience.org/library/papers/17709481PubMed
830,559,744B Cell Siglecs-News on Signaling and Its Interplay With Ligand Binding.Front Immunol2018https://conductscience.org/library/papers/30559744PubMed
912,486,104Non-T cell activation linker (NTAL): a transmembrane adaptor protein involved in immunoreceptor signaling.J Exp Med2002https://conductscience.org/library/papers/12486104PubMed
109,480,991Inhibition of the B cell by CD22: a requirement for Lyn.J Exp Med1998https://conductscience.org/library/papers/9480991PubMed

Cited by0

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Genes0

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None in this release.

Drugs0

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None in this release.

Data0

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None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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