Paper · 48197

Human lymphocytes recognise mouse alloantigens.

Lindahl KF and 1 other. Nature, 1975. Cited by 77 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Human lymphocytes recognise mouse alloantigens. Papers, record 48197. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/48197

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Human lymphocytes recognise mouse alloantigens.; PubMed
Journal: Nature; PubMed
Year: 1975; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/48197/; PubMed
DOI: https://doi.org/10.1038/254607a0; PubMed
The journal: https://conductscience.org/library/journal-index/0410462; PubMed
Citations, by iCite: 77; iCite
Relative citation ratio, by iCite: 1.73; iCite
Revised in PubMed: 2026-10-10; PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Lindahl KF	K F
2	Bach FH	F H

Source PubMed

Topics9

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Histocompatibility Antigens	Major topic
Lymphocytes	Major topic
Animals
Cytotoxicity Tests, Immunologic
Epitopes
Genotype
Humans
Mice
Species Specificity

Source PubMed

Grants0

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Trials0

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Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References15

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	4,267,208	Cell-mediated lympholysis. Importance of serologically defined H-2 regions.	J Exp Med	1973	https://conductscience.org/library/papers/4267208	PubMed
2	5,656,875	Graft versus host reaction in tissue culture. I. Lysis of monolayers of embryo mouse cells from strains differing in the H-2 histocompatibility locus by rat lymphocytes sensitized in vitro.	Immunology	1968	https://conductscience.org/library/papers/5656875	PubMed
3	4,588,912	Elicitation of selective T and B lymphocyte responses by cell surface binding ligands.	Transplant Rev	1972	https://conductscience.org/library/papers/4588912	PubMed
4	4,761,631	Effects of hydrocortisone treatment and whole body irradiation on mouse lymphocyte stimulation in vitro.	Immunology	1973	https://conductscience.org/library/papers/4761631	PubMed
5	4,140,238	Effect of antigen density on complement-mediated lysis, T-cell-mediated killing, and antigenic modulation.	J Natl Cancer Inst	1974	https://conductscience.org/library/papers/4140238	PubMed
6	4,547,153	Cell-mediated cytotoxicity to trinitrophenyl-modified syngeneic lymphocytes.	Eur J Immunol	1974	https://conductscience.org/library/papers/4547153	PubMed
7	4,179,068	Isolation of mononuclear cells and granulocytes from human blood. Isolation of monuclear cells by one centrifugation, and of granulocytes by combining centrifugation and sedimentation at 1 g.	Scand J Clin Lab Invest Suppl	1968	https://conductscience.org/library/papers/4179068	PubMed
8	5,938,778	One-way stimulation in mixed leukocyte cultures.	Science	1966	https://conductscience.org/library/papers/5938778	PubMed
9	4,139,231	Genetic control of cell-mediated lympholysis in mouse.	J Exp Med	1974	https://conductscience.org/library/papers/4139231	PubMed
10	4,398,839	In vitro induction of a heterograft reaction: immunological parameters of the sensitization of rat lymphocytes against mouse cells in vitro.	Transplantation	1971	https://conductscience.org/library/papers/4398839	PubMed

1 to 10 of 15

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Cited by15

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 77 citations.

PMID	Title	Journal	Year	In Papers	Source
8,145,040	Human CD4 restores normal T cell development and function in mice deficient in murine CD4.	J Exp Med	1994	https://conductscience.org/library/papers/8145040	PubMed
6,972,043	T cells respond preferentially to antigens that are similar to self H-2.	Proc Natl Acad Sci U S A	1981	https://conductscience.org/library/papers/6972043	PubMed
6,196,283	Xenogeneic cytotoxic T-cell clones recognize alloantigenic determinants on HLA-A2.	Immunogenetics	1983	https://conductscience.org/library/papers/6196283	PubMed
3,892,536	Human cytotoxic T-lymphocyte recognition of an HLA-A3 gene product expressed on murine L cells: the only human gene product required on the target cells for lysis is the class I heavy chain.	Proc Natl Acad Sci U S A	1985	https://conductscience.org/library/papers/3892536	PubMed
2,553,516	Transfected rat islet tumour cells express mouse major histocompatibility complex class I antigens functionally. Applicable as "pseudo-syngeneic" targets in the multiple low-dose streptozotocin diabetes model.	Diabetologia	1989	https://conductscience.org/library/papers/2553516	PubMed
1,534,116	Species specificity and augmentation of responses to class II major histocompatibility complex molecules in human CD4 transgenic mice.	J Exp Med	1992	https://conductscience.org/library/papers/1534116	PubMed
1,108,012	Functional interactions of viral and histocompatibility antigens at tumor cell surfaces.	Proc Natl Acad Sci U S A	1975	https://conductscience.org/library/papers/1108012	PubMed
1,086,476	Cells that express viral antigens but lack H-2 determinants are not lysed by immune thymus-derived lymphocytes but are lysed by other antiviral immune attack mechanisms.	Proc Natl Acad Sci U S A	1976	https://conductscience.org/library/papers/1086476	PubMed
1,082,492	Participation of the H-2 antigens of tumor cells in their lysis by syngeneic T cells.	J Exp Med	1976	https://conductscience.org/library/papers/1082492	PubMed
310,125	Induction of secondary cytotoxic T lymphocytes by purified HLA-A and HLA-B antigens reconstituted into phospholipid vesicles.	Proc Natl Acad Sci U S A	1978	https://conductscience.org/library/papers/310125	PubMed

1 to 10 of 15

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Drugs0

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Data0

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Data dates

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PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

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ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

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ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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