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Paper5409477

Regenerative calcium release within muscle cells.

Ford LE and 1 other. Science, 1970. Cited by 375 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Regenerative calcium release within muscle cells. Papers, record 5409477. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/5409477

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Regenerative calcium release within muscle cells.
PubMed
Journal
Science
PubMed
Year
1970
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
375
iCite
Relative citation ratio, by iCite
8.85
iCite
Revised in PubMed
2019-06-18
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Ford LEL E
2Podolsky RJR J

Source PubMed

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
CalciumMajor topic
MyofibrilsMajor topic
RegenerationMajor topic
Animals
Anura
Chelating Agents
In Vitro Techniques
Magnesium
Membrane Potentials
Muscle Contraction

Source PubMed

Cited by123

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 375 citations.

PMIDTitleJournalYearIn PapersSource
41,993,147Autophagy selectively clears ER in TNF-α-induced muscle atrophy.Autophagy Rep2026https://conductscience.org/library/papers/41993147PubMed
41,824,801Ca2+ signalling in cardiac muscle: the importance of balances.Europace2026https://conductscience.org/library/papers/41824801PubMed
40,838,888RyR1-mediated Ca2+-induced Ca2+ release plays a negligible role in excitation-contraction coupling of normal skeletal muscle.Proc Natl Acad Sci U S A2025https://conductscience.org/library/papers/40838888PubMed
40,563,532Calcium Signaling Dynamics in Vascular Cells and Their Dysregulation in Vascular Disease.Biomolecules2025https://conductscience.org/library/papers/40563532PubMed
39,565,684Role of ryanodine receptor cooperativity in Ca2+-wave-mediated triggered activity in cardiomyocytes.J Physiol2024https://conductscience.org/library/papers/39565684PubMed
39,220,738New evidence supports RYR3 as a candidate gene for developmental and epileptic encephalopathy.Front Neurol2024https://conductscience.org/library/papers/39220738PubMed
36,589,284The spatio-temporal properties of calcium transients in hippocampal pyramidal neurons in vitro.Front Cell Neurosci2022https://conductscience.org/library/papers/36589284PubMed
32,040,690Regulatory mechanisms of ryanodine receptor/Ca2+ release channel revealed by recent advancements in structural studies.J Muscle Res Cell Motil2021https://conductscience.org/library/papers/32040690PubMed
31,878,335Ca2+ Channels Mediate Bidirectional Signaling between Sarcolemma and Sarcoplasmic Reticulum in Muscle Cells.Cells2019https://conductscience.org/library/papers/31878335PubMed
29,514,865Calcium-induced release of calcium in muscle: 50 years of work and the emerging consensus.J Gen Physiol2018https://conductscience.org/library/papers/29514865PubMed
29,452,094Evolving mechanisms of vascular smooth muscle contraction highlight key targets in vascular disease.Biochem Pharmacol2018https://conductscience.org/library/papers/29452094PubMed
28,510,104Historical perspective on heart function: the Frank-Starling Law.Biophys Rev2015https://conductscience.org/library/papers/28510104PubMed
26,643,865Membrane depolarization increases ryanodine sensitivity to Ca2+ release to the cytosol in L6 skeletal muscle cells: Implications for excitation-contraction coupling.Exp Biol Med (Maywood)2016https://conductscience.org/library/papers/26643865PubMed
24,987,008Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Physiol Rev2014https://conductscience.org/library/papers/24987008PubMed
24,327,840Neuronal activity-regulated gene transcription: how are distant synaptic signals conveyed to the nucleus?F1000Res2012https://conductscience.org/library/papers/24327840PubMed
23,220,129Ca²⁺ waves in the heart.J Mol Cell Cardiol2013https://conductscience.org/library/papers/23220129PubMed
22,310,315Synthetic localized calcium transients directly probe signalling mechanisms in skeletal muscle.J Physiol2012https://conductscience.org/library/papers/22310315PubMed
22,155,929Longitudinal and transversal propagation of excitation along the tubular system of rat fast-twitch muscle fibres studied by high speed confocal microscopy.J Physiol2012https://conductscience.org/library/papers/22155929PubMed
22,021,327The interaction of Ca2+ with sarcomeric proteins: role in function and dysfunction of the heart.Am J Physiol Heart Circ Physiol2012https://conductscience.org/library/papers/22021327PubMed
20,228,624Spatiotemporal dynamics of Ca2+ signaling and its physiological roles.Proc Jpn Acad Ser B Phys Biol Sci2010https://conductscience.org/library/papers/20228624PubMed

See all 123 cited by

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
  • Trials
  • Comments and corrections
  • References
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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