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Paper · 62017

Suppression by autogenous complementary idiotypes: the priority of the first response.

Rowley DA and 4 others. J Exp Med, 1976. Cited by 46 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Suppression by autogenous complementary idiotypes: the priority of the first response. Papers, record 62017. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/62017

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Suppression by autogenous complementary idiotypes: the priority of the first response.
PubMed
Journal
J Exp Med
PubMed
Year
1976
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
46
iCite
Relative citation ratio, by iCite
1.02
iCite
Revised in PubMed
2019-05-08
PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Rowley DAD A
2Köhler HH
3Schreiber HH
4Kaye STS T
5Lorbach II

Source PubMed

Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Antibodies, Anti-IdiotypicMajor topic
EpitopesMajor topic
Myeloma ProteinsMajor topic
PlasmacytomaMajor topic
Animals
Antibody Specificity
Antibody-Producing Cells
Cross Reactions
Female
Immunoglobulin M
Lymph Nodes
Mice
Mice, Inbred A
Mice, Inbred BALB C
Spleen

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References31

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
115,776,574Quantitative investigations of idiotypic antibodies. V. Factors affecting the persistence and replacement of clones of antibody-producing cells.J Exp Med1971https://conductscience.org/library/papers/15776574PubMed
253,257Induction of antiphosphorylcholine antibody formation by anti-idiotypic antibodies.J Exp Med1975https://conductscience.org/library/papers/53257PubMed
34,109,640Antigen-binding myeloma proteins in mice.Ann N Y Acad Sci1971https://conductscience.org/library/papers/4109640PubMed
44,113,485Specific inhibition of plaque formation to phosphorylcholine by antibody against antibody.Science1972https://conductscience.org/library/papers/4113485PubMed
54,115,135Idiotypic determinants of immunoglobulin M detected on the surface of human lymphocytes by cytotoxicity assays.J Exp Med1972https://conductscience.org/library/papers/4115135PubMed
64,125,439Specific suppression of immune responses.Science1973https://conductscience.org/library/papers/4125439PubMed
74,128,448Clonal nature of the immune response to phosphorylcholine. I. Specificity, class, and idiotype of phosphorylcholine-binding receptors on lymphoid cells.J Exp Med1974https://conductscience.org/library/papers/4128448PubMed
84,130,243Studies of idiotypic antibodies. Production and characterization of autoantiidiotypic antisera.J Exp Med1974https://conductscience.org/library/papers/4130243PubMed
94,131,516Genetics of a new IgVH (T15 idiotype) marker in the mouse regulating natural antibody to phosphorylcholine.J Exp Med1974https://conductscience.org/library/papers/4131516PubMed
104,137,581Clonal nature of the immune response to phosphorylcholine. IV. Idiotypic uniformity of binding site-associated antigenic determinants among mouse antiphosphorylcholine antibodies.J Exp Med1974https://conductscience.org/library/papers/4137581PubMed

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Cited by15

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 46 citations.

PMIDTitleJournalYearIn PapersSource
8,350,057A first or dominant immunization. I. Suppression of simultaneous cytolytic T cell responses to unrelated alloantigens.J Exp Med1993https://conductscience.org/library/papers/8350057PubMed
7,153,709Anti-phosphorylcholine antibodies of the T15 idiotype are optimally protective against Streptococcus pneumoniae.J Exp Med1982https://conductscience.org/library/papers/7153709PubMed
7,108,445Deregulation of idiotype expression. Induction of tolerance in an anti-idiotypic response.J Exp Med1982https://conductscience.org/library/papers/7108445PubMed
7,019,373Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VII. Idiotype-specific suppression of plaque-forming cell responses.J Exp Med1981https://conductscience.org/library/papers/7019373PubMed
6,972,994Generation of idiotype-specific T cell help through network perturbation.J Exp Med1981https://conductscience.org/library/papers/6972994PubMed
6,454,749Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. IX. Characterization of Idiotype-specific effector-phase suppressor cells on plaque-forming cell responses in vitro.J Exp Med1981https://conductscience.org/library/papers/6454749PubMed
6,454,747Regulation by complementary idiotypes. Ig protects the clone producing it.J Exp Med1981https://conductscience.org/library/papers/6454747PubMed
6,333,943Induction of anti-DNA autoanti-idiotypic antibodies in (NZB X NZW)F1 mice: possible role for specific immune suppression.Clin Exp Immunol1984https://conductscience.org/library/papers/6333943PubMed
312,903
Production of auto-anti-idiotypic antibody during the normal immune response to TNP-ficoll. I. Occurrence in AKR/J and BALB/c mice of hapten-augmentable, anti-TNP plaque-forming cells and their accelerated appearance in recipients of immune spleen cells.
J Exp Med1979https://conductscience.org/library/papers/312903PubMed
307,766Use of anti-idiotypic antibodies as cell-surface receptor probes.Proc Natl Acad Sci U S A1978https://conductscience.org/library/papers/307766PubMed

1 to 10 of 15

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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