Paper · 6397403
Molecular cloning of the GAL80 gene from Saccharomyces cerevisiae and characterization of a gal80 deletion.
Yocum RR and 1 other. Gene, 1984. Cited by 46 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Molecular cloning of the GAL80 gene from Saccharomyces cerevisiae and characterization of a gal80 deletion. Papers, record 6397403. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6397403
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Molecular cloning of the GAL80 gene from Saccharomyces cerevisiae and characterization of a gal80 deletion.
- PubMed
Topics11
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Fungal Proteins | Major topic |
| Genes, Fungal | Major topic |
| Genes, Regulator | Major topic |
| Repressor Proteins | Major topic |
| Saccharomyces cerevisiae | Major topic |
| Transcription Factors | Major topic |
| Chromosome Deletion | |
| Gene Expression Regulation | |
| Glucose | |
| Operon | |
| Transformation, Genetic |
Source PubMed
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Trials0
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Comments and corrections0
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References0
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Cited by30
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 46 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 23,852,385 | A new system for comparative functional genomics of Saccharomyces yeasts. | Genetics | 2013 | https://conductscience.org/library/papers/23852385 | PubMed |
| 19,651,897 | Gene activation by dissociation of an inhibitor from a transcriptional activation domain. | Mol Cell Biol | 2009 | https://conductscience.org/library/papers/19651897 | PubMed |
| 18,456,892 | Synaptic transmission in neurons that express the Drosophila atypical soluble guanylyl cyclases, Gyc-89Da and Gyc-89Db, is necessary for the successful completion of larval and adult ecdysis. | J Exp Biol | 2008 | https://conductscience.org/library/papers/18456892 | PubMed |
| 16,219,783 | Intragenic suppression of Gal3C interaction with Gal80 in the Saccharomyces cerevisiae GAL gene switch. | Genetics | 2006 | https://conductscience.org/library/papers/16219783 | PubMed |
| 15,695,361 | Gal80 dimerization and the yeast GAL gene switch. | Genetics | 2005 | https://conductscience.org/library/papers/15695361 | PubMed |
| 10,866,670 | Evidence for Gal3p's cytoplasmic location and Gal80p's dual cytoplasmic-nuclear location implicates new mechanisms for controlling Gal4p activity in Saccharomyces cerevisiae. | Mol Cell Biol | 2000 | https://conductscience.org/library/papers/10866670 | PubMed |
| 10,523,671 | The Gal3p-Gal80p-Gal4p transcription switch of yeast: Gal3p destabilizes the Gal80p-Gal4p complex in response to galactose and ATP. | Mol Cell Biol | 1999 | https://conductscience.org/library/papers/10523671 | PubMed |
| 8,441,621 | Glucose repression of lactose/galactose metabolism in Kluyveromyces lactis is determined by the concentration of the transcriptional activator LAC9 (K1GAL4) [corrected]. | Nucleic Acids Res | 1993 | https://conductscience.org/library/papers/8441621 | PubMed |
| 8,285,714 | Fermentation of lactose by yeast cells secreting recombinant fungal lactase. | Appl Environ Microbiol | 1993 | https://conductscience.org/library/papers/8285714 | PubMed |
| 3,540,605 | Effects of null mutations in the hexokinase genes of Saccharomyces cerevisiae on catabolite repression. | Mol Cell Biol | 1986 | https://conductscience.org/library/papers/3540605 | PubMed |
1 to 10 of 30
See all 30 cited byGenes0
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None in this release.
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None in this release.
Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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