Paper · 6965523
Muscarinic suppression of a novel voltage-sensitive K+ current in a vertebrate neurone.
Brown DA and 1 other. Nature, 1980. Cited by 1,190 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Muscarinic suppression of a novel voltage-sensitive K+ current in a vertebrate neurone. Papers, record 6965523. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6965523
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics12
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Ganglia, Sympathetic | Major topic |
| Potassium | Major topic |
| Receptors, Cholinergic | Major topic |
| Receptors, Muscarinic | Major topic |
| Animals | |
| Anura | |
| Electric Conductivity | |
| In Vitro Techniques | |
| Muscarine | |
| Neurons | |
| Rana catesbeiana | |
| Tetraethylammonium Compounds |
Source PubMed
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References0
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Cited by587
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 1,190 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,776,753 | Voltage-sensor conformational microenvironments encode isoform-selective ion channel activation. | Proc Natl Acad Sci U S A | 2026 | https://conductscience.org/library/papers/42776753 | PubMed |
| 42,717,828 | Physiological 17β-oestradiol inhibits KV7.4 currents via GPER1-dependent PLC/PKC signalling. | Channels (Austin) | 2026 | https://conductscience.org/library/papers/42717828 | PubMed |
| 42,631,357 | Molecular Basis for Activation to Inhibition Switching in Kv7.2 Channel Modulators. | Angew Chem Int Ed Engl | 2026 | https://conductscience.org/library/papers/42631357 | PubMed |
| 42,571,115 | Acetylcholine enhances deviance detection in Hodgkin-Huxley neuronal networks. | Cogn Neurodyn | 2026 | https://conductscience.org/library/papers/42571115 | PubMed |
| 42,503,432 | μ-Opioid receptor signalling enhances Kir3 currents in glutamatergic preBötzinger complex neurons in mice. | J Physiol | 2026 | https://conductscience.org/library/papers/42503432 | PubMed |
| 42,295,026 | Cholesterol and Cholesterol-Derived Molecules Differentially Modulate Neuronal Kv7.2/7.3 Channels. | Arch Pharm (Weinheim) | 2026 | https://conductscience.org/library/papers/42295026 | PubMed |
| 42,261,628 | Channel Dysfunction as the Basis for Comorbidities in Multiple Sclerosis and Depression. | Arch Pharm (Weinheim) | 2026 | https://conductscience.org/library/papers/42261628 | PubMed |
| 42,261,627 | Kv7 and K2P Potassium Channels in Thalamocortical Function and Their Therapeutic Potential in Childhood Absence Epilepsy. | Arch Pharm (Weinheim) | 2026 | https://conductscience.org/library/papers/42261627 | PubMed |
| 42,198,387 | Evidence for Potentiation of M-Type Potassium Current by Flavonoid Corylin (3-(2,2-Dimethylchromen-6-yl)-7-hydroxychromen-4-one). | Pharmaceuticals (Basel) | 2026 | https://conductscience.org/library/papers/42198387 | PubMed |
| 42,136,727 | A novel image-based neuronal network model framework for understanding visual multistability and neurological disorders. | Front Comput Neurosci | 2026 | https://conductscience.org/library/papers/42136727 | PubMed |
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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