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Paper · 6970525

The role of T cell sets in the rejection of a methylcholanthrene-induced sarcoma (S1509a) in syngeneic mice.

Bhan AK and 5 others. Am J Pathol, 1981. Cited by 85 papers, by iCite.

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ConductScience Library. The role of T cell sets in the rejection of a methylcholanthrene-induced sarcoma (S1509a) in syngeneic mice. Papers, record 6970525. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6970525

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
The role of T cell sets in the rejection of a methylcholanthrene-induced sarcoma (S1509a) in syngeneic mice.
PubMed
Journal
Am J Pathol
PubMed
Year
1981
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
85
iCite
Relative citation ratio, by iCite
2.56
iCite
Revised in PubMed
2018-11-13
PubMed

Authors6

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Bhan AKA K
2Perry LLL L
3Cantor HH
4McCluskey RTR T
5Benacerraf BB
6Greene MIM I

Source PubMed

Topics9

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Graft RejectionMajor topic
Sarcoma, ExperimentalMajor topic
T-LymphocytesMajor topic
Animals
Antigens, Surface
Female
Methylcholanthrene
Mice
Neoplasm Transplantation

Source PubMed

Grants4

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
5 PO1 CA-14723NCI NIH HHSUnited StatesP01CA014723PubMed and NIHNIH RePORTER
CA-09141-03NCI NIH HHSUnited StatesPubMedPubMed
HL 18646NHLBI NIH HHSUnited StatesP01HL018646PubMed and NIHNIH RePORTER
T32CA009141NIHNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References24

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
14,587,740A rapid method for the isolation of functional thymus-derived murine lymphocytes.Eur J Immunol1973https://conductscience.org/library/papers/4587740PubMed
2234,178Ly antigens as markers for functionally distinct subpopulations of thymus-derived lymphocytes of the mouse.Nature1975https://conductscience.org/library/papers/234178PubMed
31,092,798Functional subclasses of T-lymphocytes bearing different Ly antigens. I. The generation of functionally distinct T-cell subclasses is a differentiative process independent of antigen.J Exp Med1975https://conductscience.org/library/papers/1092798PubMed
41,092,799Functional subclasses of T lymphocytes bearing different Ly antigens. II. Cooperation between subclasses of Ly+ cells in the generation of killer activity.J Exp Med1975https://conductscience.org/library/papers/1092799PubMed
51,254,955Regulation of the immune response to tumor antigens. II. The nature of immunosuppressor cells in tumor-bearing hosts.J Immunol1976https://conductscience.org/library/papers/1254955PubMed
61,083,887
Separation of helper T cells from suppressor T cells expressing different Ly components. I. Polyclonal activation: suppressor and helper activities are inherent properties of distinct T-cell subclasses.
J Exp Med1976https://conductscience.org/library/papers/1083887PubMed
71,083,888
Separation of helper T cells from suppressor T cells expressing different Ly components. II. Activation by antigen: after immunization, antigen-specific suppressor and helper activities are mediated by distinct T-cell subclasses.
J Exp Med1976https://conductscience.org/library/papers/1083888PubMed
81,083,891Cell-mediated immunity: delayed-type hypersensitivity and cytotoxic responses are mediated by different T-cell subclasses.J Exp Med1976https://conductscience.org/library/papers/1083891PubMed
91,084,399Ly and Ia antigen phenotypes of T cells involved in delayed-type hypersensitivity and in suppression.J Exp Med1976https://conductscience.org/library/papers/1084399PubMed
101,085,418Separation of suppressor and killer T cells by surgace phenotype.Nature1976https://conductscience.org/library/papers/1085418PubMed

1 to 10 of 24

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Cited by12

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 85 citations.

PMIDTitleJournalYearIn PapersSource
8,726,179Immunological memory induced by genetically transduced tumor cells.Ann Surg Oncol1996https://conductscience.org/library/papers/8726179PubMed
7,046,454Radiation-induced augmentation of the response of A/J mice to SaI tumor cells.Am J Pathol1982https://conductscience.org/library/papers/7046454PubMed
7,044,130Cell surface phenotypes of radiolabeled immune long-lived lymphocytes that selectively localize in syngeneic tumours.Am J Pathol1982https://conductscience.org/library/papers/7044130PubMed
6,609,361Malignant growth in the normal host after variant selection in vitro with cytolytic T-cell lines.Proc Natl Acad Sci U S A1984https://conductscience.org/library/papers/6609361PubMed
6,293,112Regulation of the immune response to antigens on the malignant cell surface.Springer Semin Immunopathol1982https://conductscience.org/library/papers/6293112PubMed
6,232,206In vitro activation of natural killer-like cytotoxicity by specifically in vivo primed T-helper lymphocytes in the rat.Immunology1984https://conductscience.org/library/papers/6232206PubMed
3,886,126Inhibition of growth and metastasis of syngeneic transplantable tumours by an aromatic retinoic acid analogue. 2. T cell dependence of retinoid effects in vivo.Cancer Immunol Immunother1985https://conductscience.org/library/papers/3886126PubMed
3,002,606Correlation of tumor specific delayed type hypersensitivity reaction and tumor protection to SV40-induced mKSA fibrosarcoma.Cancer Immunol Immunother1986https://conductscience.org/library/papers/3002606PubMed
2,974,763Helper strategy in tumor immunology: expansion of helper lymphocytes and utilization of helper lymphokines for experimental and clinical immunotherapy.Cancer Metastasis Rev1988https://conductscience.org/library/papers/2974763PubMed
2,938,736The augmentation of tumor-specific immunity by virus help. III. Enhanced generation of tumor-specific Lyt-1+2- T cells is responsible for augmented tumor immunity in vivo.Cancer Immunol Immunother1986https://conductscience.org/library/papers/2938736PubMed

1 to 10 of 12

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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