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Paper · 7084465

Decreased transport of thyroxine (T4), 3,3',5-triiodothyronine (T3) and 3,3',5'-triiodothyronine (rT3) into rat hepatocytes in primary culture due to a decrease of cellular ATP content and various drugs.

Krenning EP and 4 others. FEBS Lett, 1982. Cited by 110 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Decreased transport of thyroxine (T4), 3,3',5-triiodothyronine (T3) and 3,3',5'-triiodothyronine (rT3) into rat hepatocytes in primary culture due to a decrease of cellular ATP content and various drugs. Papers, record 7084465. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/7084465

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Decreased transport of thyroxine (T4), 3,3',5-triiodothyronine (T3) and 3,3',5'-triiodothyronine (rT3) into rat hepatocytes in primary culture due to a decrease of cellular ATP content and various drugs.
PubMed
Journal
FEBS Lett
PubMed
Year
1982
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
110
iCite
Relative citation ratio, by iCite
3.7
iCite
Revised in PubMed
2019-06-21
PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Krenning EPE P
2Docter RR
3Bernard BB
4Visser TT
5Hennemann GG

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Adenosine TriphosphateMajor topic
LiverMajor topic
ThyroxineMajor topic
TriiodothyronineMajor topic
Triiodothyronine, ReverseMajor topic
Amiodarone
Animals
Biological Transport, Active
Cells, Cultured
Colchicine
Cytochalasin B
Propranolol
Rats
Vinblastine

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by17

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 110 citations.

PMIDTitleJournalYearIn PapersSource
39,076,009Novel Biomarkers Reveal Mismatch Between Tissue and Serum Thyroid Hormone Status in Amiodarone-Induced Hyperthyroidism.J Clin Endocrinol Metab2025https://conductscience.org/library/papers/39076009PubMed
36,818,930AMIODARONE AND THYROID DYSFUNCTION.Acta Clin Croat2022https://conductscience.org/library/papers/36818930PubMed
36,376,450Developmental changes of the fetal and neonatal thyroid gland and functional consequences on the cardiovascular system.J Perinatol2022https://conductscience.org/library/papers/36376450PubMed
31,033,998Paradigms of Dynamic Control of Thyroid Hormone Signaling.Endocr Rev2019https://conductscience.org/library/papers/31033998PubMed
30,309,693Amiodarone and thyroid physiology, pathophysiology, diagnosis and management.Trends Cardiovasc Med2019https://conductscience.org/library/papers/30309693PubMed
25,477,968Amiodarone-induced thyrotoxic thyroiditis: a diagnostic and therapeutic challenge.Case Rep Med2014https://conductscience.org/library/papers/25477968PubMed
20,926,587Inhibition of the type 2 iodothyronine deiodinase underlies the elevated plasma TSH associated with amiodarone treatment.Endocrinology2010https://conductscience.org/library/papers/20926587PubMed
11,263,469Effects of amiodarone administration during pregnancy on neonatal thyroid function and subsequent neurodevelopment.J Endocrinol Invest2001https://conductscience.org/library/papers/11263469PubMed
7,159,396Evidence that the uptake of tri-iodo-L-thyronine by human erythrocytes is carrier-mediated but not energy-dependent.Biochem J1982https://conductscience.org/library/papers/7159396PubMed
6,841,918Plasma membrane transport of thyroid hormone: its possible pathophysiological significance.J Endocrinol Invest1983https://conductscience.org/library/papers/6841918PubMed

1 to 10 of 17

See all 17 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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