Paper · 73137
Evidence for an H-2/viral protein complex on the cell surface as the basis for the H-2 restriction of cytotoxicity.
Blank KJ and 1 other. Nature, 1977. Cited by 101 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Evidence for an H-2/viral protein complex on the cell surface as the basis for the H-2 restriction of cytotoxicity. Papers, record 73137. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/73137
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Evidence for an H-2/viral protein complex on the cell surface as the basis for the H-2 restriction of cytotoxicity.
- PubMed
Topics12
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Antigens, Viral | Major topic |
| Cytotoxicity, Immunologic | Major topic |
| H-2 Antigens | Major topic |
| Leukemia, Experimental | Major topic |
| T-Lymphocytes | Major topic |
| Animals | |
| Antigen-Antibody Reactions | |
| Epitopes | |
| Friend murine leukemia virus | |
| Immunization, Passive | |
| Isoantibodies | |
| Mice |
Source PubMed
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Trials0
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Comments and corrections0
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References0
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Cited by27
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 101 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 22,457,924 | Anti-H-2 antibodies induced by syngeneic immunization. | Immunogenetics | 1980 | https://conductscience.org/library/papers/22457924 | PubMed |
| 7,687,300 | Multiplicity of virus-encoded helper T-cell epitopes expressed on FBL-3 tumor cells. | J Virol | 1993 | https://conductscience.org/library/papers/7687300 | PubMed |
| 7,441,823 | Possible cell surface receptor for Friend murine leukemia virus isolated with viral envelope glycoprotein complexes. | J Virol | 1980 | https://conductscience.org/library/papers/7441823 | PubMed |
| 7,333,657 | Variation of expression of histocompatibility antigens on tumor cells: absence of H-2Kk-gene products from a gross-virus-induced leukemia in BALB.K. | Immunogenetics | 1981 | https://conductscience.org/library/papers/7333657 | PubMed |
| 7,000,703 | Independence of chicken major histocompatibility antigens and tumor-associated antigen on the surface of herpesvirus-induced lymphoma cells. | Infect Immun | 1980 | https://conductscience.org/library/papers/7000703 | PubMed |
| 6,982,849 | Resistance to cell-mediated cytotoxicity is correlated with reduction of H-2K gene products in AKR leukemia. | Immunogenetics | 1982 | https://conductscience.org/library/papers/6982849 | PubMed |
| 6,935,387 | H-2D (Rfv-1) gene influence on recovery from Friend virus leukemia is mediated by nonleukemic cells of the spleen and bone marrow. | J Exp Med | 1980 | https://conductscience.org/library/papers/6935387 | PubMed |
| 6,770,026 | Immune response genes control T killer cell response against Moloney tumor antigen cytolysis regulating reactions against the best available H-2 + viral antigen association. | J Exp Med | 1980 | https://conductscience.org/library/papers/6770026 | PubMed |
| 6,406,638 | H-2D control of recovery from Friend virus leukemia: H-2D region influences the kinetics of the T lymphocyte response to Friend virus. | J Exp Med | 1983 | https://conductscience.org/library/papers/6406638 | PubMed |
| 6,277,784 | Independence of H-2 and viral antigens on the cell surface and absence of H-2 antigens on murine leukemia virus and mouse mammary tumor virus particles. | Immunogenetics | 1981 | https://conductscience.org/library/papers/6277784 | PubMed |
1 to 10 of 27
See all 27 cited byGenes0
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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