Paper75170

Myelin basic protein: structure, function and antigenic determinants.

Hashim GA. Immunol Rev, 1978. Cited by 169 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Myelin basic protein: structure, function and antigenic determinants. Papers, record 75170. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/75170

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Myelin basic protein: structure, function and antigenic determinants.; PubMed
Journal: Immunol Rev; PubMed
Year: 1978; PubMed
Publication types: Journal Article, Research Support, U.S. Gov't, P.H.S., Review; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/75170/; PubMed
DOI: https://doi.org/10.1111/j.1600-065x.1978.tb00397.x; PubMed
The journal: https://conductscience.org/library/journal-index/7702118; PubMed
Citations, by iCite: 169; iCite
Relative citation ratio, by iCite: 4.62; iCite
Revised in PubMed: 2021-12-03; PubMed

Authors1

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Hashim GA	G A

Source PubMed

Topics20

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Epitopes	Major topic
Myelin Basic Protein	Major topic
Amino Acid Sequence
Animals
Encephalomyelitis, Autoimmune, Experimental
Guinea Pigs
Haplorhini
Humans
Hypersensitivity, Delayed
Immunity, Cellular
Immunization
Immunization, Passive
Immunosuppression Therapy
Mice
Multiple Sclerosis
Myelin Proteins
Rabbits
Rats
Rosette Formation
T-Lymphocytes

Source PubMed

Show 14 more topicsShow fewer topics

Cited by32

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 169 citations.

PMID	Title	Journal	Year	In Papers	Source
37,239,982	EAE of Mice: Enzymatic Cross Site-Specific Hydrolysis of H2A Histone by IgGs against H2A, H1, H2B, H3, and H4 Histones and Myelin Basic Protein.	Int J Mol Sci	2023	https://conductscience.org/library/papers/37239982	PubMed
37,049,736	EAE of Mice: Enzymatic Cross Site-Specific Hydrolysis of H2B Histone by IgGs against H1, H2A, H2B, H3, and H4 Histones and Myelin Basic Protein.	Molecules	2023	https://conductscience.org/library/papers/37049736	PubMed
36,012,448	Experimental Autoimmune Encephalomyelitis of Mice: Enzymatic Cross Site-Specific Hydrolysis of H4 Histone by IgGs against Histones and Myelin Basic Protein.	Int J Mol Sci	2022	https://conductscience.org/library/papers/36012448	PubMed
15,067,168	Probable epitopes: Relationships between myelin basic protein antigenic determinants and viral and bacterial proteins.	Neuroinformatics	2004	https://conductscience.org/library/papers/15067168	PubMed
10,502,543	Neuroimmunotoxicology: humoral assessment of neurotoxicity and autoimmune mechanisms.	Environ Health Perspect	1999	https://conductscience.org/library/papers/10502543	PubMed
7,543,135	Prevention of experimental allergic encephalomyelitis in rats by targeting autoantigen to B cells: evidence that the protective mechanism depends on changes in the cytokine response and migratory properties of the autoantigen-specific T cells.	J Exp Med	1995	https://conductscience.org/library/papers/7543135	PubMed
7,536,156	Lead alters the immunogenicity of two neural proteins: a potential mechanism for the progression of lead-induced neurotoxicity.	Environ Health Perspect	1994	https://conductscience.org/library/papers/7536156	PubMed
6,806,429	Acute autoimmune encephalomyelitis in mice. II. Susceptibility is controlled by the combination of H-2 and histamine sensitization genes.	J Exp Med	1982	https://conductscience.org/library/papers/6806429	PubMed
6,766,340	Suppression of multiple sclerosis.	Br Med J	1980	https://conductscience.org/library/papers/6766340	PubMed
6,209,588	Immunochemical cross-reactivity between intact purified myelin basic protein (MBP) and the synthetic encephalitogenic peptide S49.	Neurochem Res	1984	https://conductscience.org/library/papers/6209588	PubMed
6,196,105	Coexistence of immunogenic and suppressogenic epitopes in tumor cells and various types of macromolecules.	Cancer Immunol Immunother	1983	https://conductscience.org/library/papers/6196105	PubMed
6,194,137	Myelin basic protein and multiple sclerosis.	Ital J Neurol Sci	1983	https://conductscience.org/library/papers/6194137	PubMed
6,177,329	Cellular immunity to encephalitogenic peptide in tumour-bearing mice.	Br J Cancer	1982	https://conductscience.org/library/papers/6177329	PubMed
6,171,742	Synthetic peptides from region 65-84 of bovine myelin basic protein: radioimmunoassays and equilibrium competitive inhibition studies with antibodies prepared against myelin basic protein.	Neurochem Res	1981	https://conductscience.org/library/papers/6171742	PubMed
6,168,926	Equilibrium and nonequilibrium competitive inhibitions of antipeptide antibody binding by parent myelin basic protein and 18 related peptide sequences.	Neurochem Res	1981	https://conductscience.org/library/papers/6168926	PubMed
6,168,025	Function of macrophages as antigen presenting cells.	Springer Semin Immunopathol	1980	https://conductscience.org/library/papers/6168025	PubMed
6,154,252	Failure of myelin basic protein to prevent or suppress experimental allergic encephalomyelitis in guinea pigs.	Neurochem Res	1980	https://conductscience.org/library/papers/6154252	PubMed
6,083,474	Interaction between human myelin basic protein and lipophilin.	Neurochem Res	1984	https://conductscience.org/library/papers/6083474	PubMed
6,083,472	Delayed-type hypersensitivity to myelin basic proteins in mice susceptible to allergic encephalomyelitis.	Neurochem Res	1984	https://conductscience.org/library/papers/6083472	PubMed
6,083,470	Format determinants of synthetic myelin basic protein peptide S82 mimicked by a mixture of synthetic peptides S8 and S79.	Neurochem Res	1984	https://conductscience.org/library/papers/6083470	PubMed

Show 14 more cited byShow fewer cited bySee all 32 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
MBP	4,155	PubTator's text mining	https://conductscience.org/library/genes/4155

Source NCBI Gene

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

None in this release

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Trials
Comments and corrections
References
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Data

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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