Paper80299
Natural killer cells in the mouse: an alternative immune surveillance mechanism?
Kiessling R and 1 other. Contemp Top Immunobiol, 1978. Cited by 132 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Natural killer cells in the mouse: an alternative immune surveillance mechanism? Papers, record 80299. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/80299
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics15
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Immunity, Cellular | Major topic |
| Killer Cells, Natural | Major topic |
| Aging | |
| Animals | |
| Antibody-Dependent Cell Cytotoxicity | |
| B-Lymphocytes | |
| Epitopes | |
| Genes | |
| Helix, Snails | |
| Hemagglutinins | |
| Macrophages | |
| Mice | |
| Monocytes | |
| Neoplasms, Experimental | |
| T-Lymphocytes |
Source PubMed
Cited by49
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 132 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 26,228,090 | Activation of protein kinase C and protein kinase D in human natural killer cells: effects of tributyltin, dibutyltin, and tetrabromobisphenol A. | Toxicol Mech Methods | 2015 | https://conductscience.org/library/papers/26228090 | PubMed |
| 23,047,847 | Tributyltin and dibutyltin alter secretion of tumor necrosis factor alpha from human natural killer cells and a mixture of T cells and natural killer cells. | J Appl Toxicol | 2013 | https://conductscience.org/library/papers/23047847 | PubMed |
| 22,177,321 | Review: Immunogenetics of human placentation. | Placenta | 2012 | https://conductscience.org/library/papers/22177321 | PubMed |
| 21,623,697 | Tetrabromobisphenol A decreases cell-surface proteins involved in human natural killer (NK) cell-dependent target cell lysis. | J Immunotoxicol | 2011 | https://conductscience.org/library/papers/21623697 | PubMed |
| 20,883,105 | Activation of p44/42 in human natural killer cells decreases cell-surface protein expression: Relationship to tributyltin-induced alterations of protein expression. | Toxicol Mech Methods | 2010 | https://conductscience.org/library/papers/20883105 | PubMed |
| 20,370,538 | Effects of butyltin exposures on MAP kinase-dependent transcription regulators in human natural killer cells. | Toxicol Mech Methods | 2010 | https://conductscience.org/library/papers/20370538 | PubMed |
| 20,297,919 | Immunosuppressive effects of triclosan, nonylphenol, and DDT on human natural killer cells in vitro. | J Immunotoxicol | 2010 | https://conductscience.org/library/papers/20297919 | PubMed |
| 20,213,532 | Activation of p44/42 MAPK plays a role in the TBT-induced loss of human natural killer (NK) cell function. | Cell Biol Toxicol | 2010 | https://conductscience.org/library/papers/20213532 | PubMed |
| 19,122,738 | Effects of a series of triorganotins on ATP levels in human natural killer cells. | Environ Toxicol Pharmacol | 2008 | https://conductscience.org/library/papers/19122738 | PubMed |
| 18,821,099 | Glutathione diminishes tributyltin- and dibutyltin-induced loss of lytic function in human natural killer cells. | Drug Chem Toxicol | 2009 | https://conductscience.org/library/papers/18821099 | PubMed |
| 18,623,605 | Pentachlorophenol decreases ATP levels in human natural killer cells. | J Appl Toxicol | 2008 | https://conductscience.org/library/papers/18623605 | PubMed |
| 18,441,343 | Serum supplementation modulates the effects of dibutyltin on human natural killer cell function. | Toxicol Sci | 2008 | https://conductscience.org/library/papers/18441343 | PubMed |
| 15,580,257 | An innately interesting decade of research in immunology. | Nat Med | 2004 | https://conductscience.org/library/papers/15580257 | PubMed |
| 9,640,254 | Lysis-sensitive targets stimulate an elevation of cAMP in human natural killer cells. | Immunology | 1998 | https://conductscience.org/library/papers/9640254 | PubMed |
| 9,536,128 | Monoclonal antibody anti-NC-2 identifies a second receptor on cells mediating natural cytotoxicity in mice. | Immunology | 1998 | https://conductscience.org/library/papers/9536128 | PubMed |
| 7,459,234 | Inhibition of cell proliferation rather than of cell lysis as a measure of immune reactivity in embryo-antigen-challenged mice. | Br J Cancer | 1981 | https://conductscience.org/library/papers/7459234 | PubMed |
| 7,426,312 | Antibody-dependent cellular cytotoxicity in cancer patients: lack of prognostic value. | Br J Cancer | 1980 | https://conductscience.org/library/papers/7426312 | PubMed |
| 7,307,338 | In vitro augmentation of human natural cytotoxic activity. | Clin Exp Immunol | 1981 | https://conductscience.org/library/papers/7307338 | PubMed |
| 7,272,185 | Mice treated with strontium 90: an animal model deficient in NK cells. | Br J Cancer | 1981 | https://conductscience.org/library/papers/7272185 | PubMed |
| 7,203,558 | Activation of natural killer (NK) cells in vivo with H-2 and non-H-2 alloantigens. | Immunogenetics | 1981 | https://conductscience.org/library/papers/7203558 | PubMed |
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
None in this release
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- Trials
- Comments and corrections
- References
- Genes
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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