Paper · 8288123
bHLH factors in muscle development: dead lines and commitments, what to leave in and what to leave out.
Olson EN and 1 other. Genes Dev, 1994. Cited by 645 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. bHLH factors in muscle development: dead lines and commitments, what to leave in and what to leave out. Papers, record 8288123. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/8288123
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics5
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| DNA-Binding Proteins | Major topic |
| Helix-Loop-Helix Motifs | Major topic |
| Muscles | Major topic |
| Animals | |
| Humans |
Source PubMed
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References0
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Cited by136
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 645 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,743,735 | Ubiquitin-proteasome-dependent IMPDH2 degradation regulated by ANKRD9 controls myogenic differentiation in Gallus gallus. | Poult Sci | 2026 | https://conductscience.org/library/papers/42743735 | PubMed |
| 40,566,792 | Decoding complexity: tackling the challenge of how many transcription factors regulate a plant gene. | Transcription | 2025 | https://conductscience.org/library/papers/40566792 | PubMed |
| 38,540,781 | Characterization of Skeletal Muscle Regeneration Revealed a Novel Growth Network Induced by Molecular Acupuncture-like Transfection. | Biomolecules | 2024 | https://conductscience.org/library/papers/38540781 | PubMed |
| 38,206,489 | Post-transcriptional regulation of myogenic transcription factors during muscle development and pathogenesis. | J Muscle Res Cell Motil | 2024 | https://conductscience.org/library/papers/38206489 | PubMed |
| 37,282,234 | The ascidian embryo: An experimental system for studying genetic circuitry for embryonic cell specification and morphogenesis. | Dev Growth Differ | 1996 | https://conductscience.org/library/papers/37282234 | PubMed |
| 35,173,013 | Intragenic suppressors unravel the role of the SCREAM ACT-like domain for bHLH partner selectivity in stomatal development. | Proc Natl Acad Sci U S A | 2022 | https://conductscience.org/library/papers/35173013 | PubMed |
| 34,502,190 | The Phosphonate Derivative of C60 Fullerene Induces Differentiation towards the Myogenic Lineage in Human Adipose-Derived Mesenchymal Stem Cells. | Int J Mol Sci | 2021 | https://conductscience.org/library/papers/34502190 | PubMed |
| 31,106,260 | Cell Mechanics of Craniosynostosis. | ACS Biomater Sci Eng | 2017 | https://conductscience.org/library/papers/31106260 | PubMed |
| 30,777,932 | Akirin1 promotes myoblast differentiation by modulating multiple myoblast differentiation factors. | Biosci Rep | 2019 | https://conductscience.org/library/papers/30777932 | PubMed |
| 30,367,407 | Transdifferentiation of Muscle Satellite Cells to Adipose Cells Using CRISPR/Cas9-Mediated Targeting of MyoD. | Methods Mol Biol | 2019 | https://conductscience.org/library/papers/30367407 | PubMed |
1 to 10 of 136
See all 136 cited byGenes0
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None in this release.
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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