Paper83234
Recognition of idiotypes in lymphocyte interactions. I. Idiotypic selectivity in the cooperation between T and B lymphocytes.
Hetzelberger D and 1 other. Eur J Immunol, 1978. Cited by 102 papers, by iCite.
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ConductScience Library. Recognition of idiotypes in lymphocyte interactions. I. Idiotypic selectivity in the cooperation between T and B lymphocytes. Papers, record 83234. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/83234
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Recognition of idiotypes in lymphocyte interactions. I. Idiotypic selectivity in the cooperation between T and B lymphocytes.
- PubMed
Topics18
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| B-Lymphocytes | Major topic |
| Immunoglobulin Idiotypes | Major topic |
| Lymphocyte Cooperation | Major topic |
| T-Lymphocytes | Major topic |
| Animals | |
| Bacterial Vaccines | |
| Epitopes | |
| Female | |
| Genotype | |
| Immunoglobulin Variable Region | |
| Immunosuppression Therapy | |
| Male | |
| Mice | |
| Mice, Inbred A | |
| Mice, Inbred BALB C | |
| Mice, Inbred C57BL | |
| Streptococcus pyogenes | |
| Trinitrobenzenes |
Source PubMed
Cited by39
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 102 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 8,188,317 | The IgG2a antibody response to thyroglobulin is linked to the Igh locus in mouse. | Immunogenetics | 1994 | https://conductscience.org/library/papers/8188317 | PubMed |
| 7,299,344 | Expression of an idiotype (Id-460) during in vivo anti-dinitrophenyl antibody responses. III. Detection of Id-460 in normal serum that does not bind dinitrophenyl. | J Exp Med | 1981 | https://conductscience.org/library/papers/7299344 | PubMed |
| 7,252,415 | Idiotype-anti-idiotype regulation. I. Immunization with a levan-binding myeloma protein leads to the appearance of auto-anti-(anti-idiotype) antibodies and to the activation of silent clones. | J Exp Med | 1981 | https://conductscience.org/library/papers/7252415 | PubMed |
| 7,028,912 | Expression of an idiotype (Id-460) during in vivo anti-dinitrophenyl antibody responses. II. Transient idiotypic dominance. | J Exp Med | 1981 | https://conductscience.org/library/papers/7028912 | PubMed |
| 6,978,921 | Helper T cell recognition of the variable domains of a mouse myeloma protein (315). Effect of the major histocompatibility complex and domain conformation. | J Exp Med | 1982 | https://conductscience.org/library/papers/6978921 | PubMed |
| 6,977,609 | Two Ly-2 T helper cell subsets distinguished by Qa-1 phenotype. The priming environment determines whether one or both subsets will be generated. | J Exp Med | 1982 | https://conductscience.org/library/papers/6977609 | PubMed |
| 6,973,603 | Antigen-specific helper T cells required for dominant idiotype expression are not H-2 restricted. | J Exp Med | 1981 | https://conductscience.org/library/papers/6973603 | PubMed |
| 6,969,777 | T cell regulation of IgG subclass antibody production in response to T-independent antigens. | J Exp Med | 1981 | https://conductscience.org/library/papers/6969777 | PubMed |
| 6,966,320 | Selective suppression of the major idiotypic component of an antihapten response by soluble T cell-derived factors with idiotypic or anti-idiotypic receptors. | J Exp Med | 1980 | https://conductscience.org/library/papers/6966320 | PubMed |
| 6,854,211 | Regulation of natural antiallotype antibody responses by idiotype network-induced auto-antiidiotypic antibodies. | J Exp Med | 1983 | https://conductscience.org/library/papers/6854211 | PubMed |
| 6,792,725 | How many T cells help one B cell? | Springer Semin Immunopathol | 1980 | https://conductscience.org/library/papers/6792725 | PubMed |
| 6,788,889 | Priming of T helper cells by antigen-activated B cells. B cell-primed Lyt-1+ helper cells are restricted to cooperate with B cells expressing the IgvH phenotype of the priming B cells. | J Exp Med | 1981 | https://conductscience.org/library/papers/6788889 | PubMed |
| 6,788,888 | Influence of Igh-linked gene products on the generation of T helper cells in the response to sheep erythrocytes. | J Exp Med | 1981 | https://conductscience.org/library/papers/6788888 | PubMed |
| 6,447,749 | Quantitative studies on T cell diversity. I. Determination of the precursor frequencies for two types of streptococcus A-specific helper cells in nonimmune, polyclonally activated splenic T cells. | J Exp Med | 1980 | https://conductscience.org/library/papers/6447749 | PubMed |
| 6,444,664 | Feedback suppression of the immune response in vitro. II. IgVH-restricted antibody-dependent suppression. | J Exp Med | 1980 | https://conductscience.org/library/papers/6444664 | PubMed |
| 6,444,660 | Anti-phenyltrimethylamino immunity in mice. II. L-Tyrosine-p-azophenyltrimethylammonium-induced suppressor T cells selectively inhibit the expression of B-cell clones bearing a cross-reactive idiotype. | J Exp Med | 1980 | https://conductscience.org/library/papers/6444660 | PubMed |
| 6,417,259 | Tolerance for self IG at the level of the Ly1+ T cell. | J Exp Med | 1983 | https://conductscience.org/library/papers/6417259 | PubMed |
| 6,225,199 | Network regulation among T cells; conclusions from limiting dilution experiments. | Springer Semin Immunopathol | 1983 | https://conductscience.org/library/papers/6225199 | PubMed |
| 6,198,421 | Non-dinitrophenyl-binding immunoglobulin that bears a dominant idiotype (Id460) associated with antidinitrophenyl antibody is specific for an antigen on Pasteurella pneumotropica. | J Exp Med | 1984 | https://conductscience.org/library/papers/6198421 | PubMed |
| 6,193,227 | Idiotype-specific T lymphocytes. II. capability of generation of idiotype-specific T lymphocytes is determined by corresponding VH gene. | J Exp Med | 1983 | https://conductscience.org/library/papers/6193227 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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