Paper · 8544844

Wilms' tumor protein WT1 as an ovarian transcription factor: decreases in expression during follicle development and repression of inhibin-alpha gene promoter.

Hsu SY and 5 others. Mol Endocrinol, 1995. Cited by 88 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Cite this record

ConductScience Library. Wilms' tumor protein WT1 as an ovarian transcription factor: decreases in expression during follicle development and repression of inhibin-alpha gene promoter. Papers, record 8544844. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/8544844

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Wilms' tumor protein WT1 as an ovarian transcription factor: decreases in expression during follicle development and repression of inhibin-alpha gene promoter.; PubMed
Journal: Mol Endocrinol; PubMed
Year: 1995; PubMed
Publication types: Journal Article, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/8544844/; PubMed
DOI: https://doi.org/10.1210/mend.9.10.8544844; PubMed
The journal: https://conductscience.org/library/journal-index/8801431; PubMed
Citations, by iCite: 88; iCite
Relative citation ratio, by iCite: 2.13; iCite
Revised in PubMed: 2008-11-21; PubMed

Authors6

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Hsu SY	S Y
2	Kubo M	M
3	Chun SY	S Y
4	Haluska FG	F G
5	Housman DE	D E
6	Hsueh AJ	A J

Source PubMed

Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
DNA-Binding Proteins	Major topic
Inhibins	Major topic
Ovarian Follicle	Major topic
Ovary	Major topic
Transcription Factors	Major topic
Animals
Cell Differentiation
Female
Gene Expression Regulation
In Situ Hybridization
Promoter Regions, Genetic
RNA, Messenger
Rats
Rats, Sprague-Dawley
WT1 Proteins

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
HD-13527	NICHD NIH HHS	United States	P01HD013527	PubMed and NIH

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by22

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 88 citations.

PMID	Title	Journal	Year	In Papers	Source
41,992,335	Integrated ATAC-seq and mRNA-seq analyses on granulosa cells identify key regulators of follicle selection in chickens.	J Anim Sci Biotechnol	2026	https://conductscience.org/library/papers/41992335	PubMed
40,257,231	Anti-Müllerian hormone in PCOS: Molecular regulation and emerging therapeutic strategies.	Biomol Biomed	2025	https://conductscience.org/library/papers/40257231	PubMed
39,349,682	Genome-wide association studies for pelvic organ prolapse in the Japanese population.	Commun Biol	2024	https://conductscience.org/library/papers/39349682	PubMed
38,763,964	P62 promotes FSH-induced antral follicle formation by directing degradation of ubiquitinated WT1.	Cell Mol Life Sci	2024	https://conductscience.org/library/papers/38763964	PubMed
36,742,369	A cis-regulatory lexicon of DNA motif combinations mediating cell-type-specific gene regulation.	Cell Genom	2022	https://conductscience.org/library/papers/36742369	PubMed
34,448,450	PRMT5 regulates ovarian follicle development by facilitating Wt1 translation.	Elife	2021	https://conductscience.org/library/papers/34448450	PubMed
32,945,847	Aberrant and constitutive expression of FOXL2 impairs ovarian development and functions in mice.	Biol Reprod	2020	https://conductscience.org/library/papers/32945847	PubMed
28,881,869	Female Fertility: It Takes Two to Tango.	Endocrinology	2017	https://conductscience.org/library/papers/28881869	PubMed
26,358,501	Novel WT1 Missense Mutations in Han Chinese Women with Premature Ovarian Failure.	Sci Rep	2015	https://conductscience.org/library/papers/26358501	PubMed
25,491,731	Wilms' tumor suppressor gene (WT1) suppresses apoptosis by transcriptionally downregulating BAX expression in immature rat granulosa cells.	J Ovarian Res	2014	https://conductscience.org/library/papers/25491731	PubMed

1 to 10 of 22

See all 22 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/8544844

GET https://conductscience.org/api/papers/v1/8544844/cited_by?page=2

API reference For AI assistants