Paper · 893544
The analysis of malignancy by cell fusion. VII. Cytogenetic analysis of hybrids between malignant and diploid cells and of tumours derived from them.
Jonasson J and 2 others. J Cell Sci, 1977. Cited by 209 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. The analysis of malignancy by cell fusion. VII. Cytogenetic analysis of hybrids between malignant and diploid cells and of tumours derived from them. Papers, record 893544. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/893544
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PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- The analysis of malignancy by cell fusion. VII. Cytogenetic analysis of hybrids between malignant and diploid cells and of tumours derived from them.
- PubMed
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Diploidy | Major topic |
| Neoplasms | Major topic |
| Animals | |
| Cell Fusion | |
| Clone Cells | |
| Cytogenetics | |
| Fibroblasts | |
| Hybridization, Genetic | |
| Karyotyping | |
| Lymphocytes | |
| Mice | |
| Neoplasms, Experimental | |
| Sarcoma |
Source PubMed
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References0
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Cited by40
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 209 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 35,912,015 | Mutation or not, what directly establishes a neoplastic state, namely cellular immortality and autonomy, still remains unknown and should be prioritized in our research. | J Cancer | 2022 | https://conductscience.org/library/papers/35912015 | PubMed |
| 34,576,139 | Deletion of Col15a1 Modulates the Tumour Extracellular Matrix and Leads to Increased Tumour Growth in the MMTV-PyMT Mouse Mammary Carcinoma Model. | Int J Mol Sci | 2021 | https://conductscience.org/library/papers/34576139 | PubMed |
| 24,252,922 | Cytogenetics and the major histocompatibility complex in a proliferating microcell-mediated hybrid clone. | Theor Appl Genet | 1985 | https://conductscience.org/library/papers/24252922 | PubMed |
| 24,248,084 | Influence of both coculture and BrdU on NOR activity of mouse, rat and human cells. | Theor Appl Genet | 1986 | https://conductscience.org/library/papers/24248084 | PubMed |
| 23,405,135 | Horizontal transmission of malignancy: in-vivo fusion of human lymphomas with hamster stroma produces tumors retaining human genes and lymphoid pathology. | PLoS One | 2013 | https://conductscience.org/library/papers/23405135 | PubMed |
| 22,531,369 | Tumor suppression by collagen XV is independent of the restin domain. | Matrix Biol | 2012 | https://conductscience.org/library/papers/22531369 | PubMed |
| 19,389,504 | Recurrent and nonrandom DNA copy number and chromosome alterations in Myc transgenic mouse model for hepatocellular carcinogenesis: implications for human disease. | Cancer Genet Cytogenet | 2009 | https://conductscience.org/library/papers/19389504 | PubMed |
| 18,171,981 | Complete suppression of tumor formation by high levels of basement membrane collagen. | Mol Cancer Res | 2007 | https://conductscience.org/library/papers/18171981 | PubMed |
| 12,420,221 | Mitochondrial stress-induced calcium signaling, phenotypic changes and invasive behavior in human lung carcinoma A549 cells. | Oncogene | 2002 | https://conductscience.org/library/papers/12420221 | PubMed |
| 11,986,397 | Molecular aspects of melanocytic dysplastic nevi. | J Mol Diagn | 2002 | https://conductscience.org/library/papers/11986397 | PubMed |
1 to 10 of 40
See all 40 cited byGenes0
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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