ConductScience.org, home

Cell line · CVCL_0179

Papers172

PaperJournalYearHow we knowRetractedSource
Impact of Trastuzumab and Pertuzumab on HER2 Localization and the Estrogen Receptor Cistrome in ER+/HER2+ Breast Cancer.bioRxiv2026Its Methods give the RRID and name the linePubMed
The role of autoproteolysis and mitoribosomal proteins in the regulation of the mitochondrial LACTB tumor suppressor.EMBO Rep2026Its Methods give the RRID and name the linePubMed
Targeting CD24 Activates Macrophages to Reduce Tumor Burden in Preclinical Models of Solid Tumors.Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Targetable S100-RAGE Signaling Mediates Intrinsic Trastuzumab Deruxtecan Resistance in Metastatic Breast Cancer.Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Targeted antibody-drug conjugates for rhabdomyosarcoma and other FGFR4-expressing cancers.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Distinct metabolic dependency on BCAA defines cancer stemness and malignancy in human triple-negative breast cancer.Cell Rep2026Its Methods give the RRID and name the linePubMed
Single-Cell and Multiomics Characterization of p21 in Cancer Progression and Therapeutic Sensitivity.Hum Mutat2026Its Methods give the RRID and name the linePubMed
Engineering Cathepsin S Selective Chemical Probes and Antibody-Drug Conjugates through Substrate Profiling with Unnatural Amino Acids.J Med Chem2026Its Methods give the RRID and name the linePubMed
Combined inhibition of PLK4 and PPM1D promotes cell death in cancer cells with 17q amplification.Cancer Cell Int2026Its Methods give the RRID and name the linePubMed
Hydrostatic pressure activates HIF-1α via β-catenin to promote stemness in breast cancer cells.FEBS Open Bio2026Its Methods give the RRID and name the linePubMed
Triple-Negative Breast Cancer Cells Utilize IL8 and CXCL1 to Suppress NK Cells' Function and Facilitate Cancer Metastasis.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
Cancer-associated fibroblast-derived protein S100-A11 influences the response to anti-HER2 therapies in HER2-positive breast cancer.Neoplasia2026Its Methods give the RRID and name the linePubMed
miR-3648 regulates cell functions and acts as a potential biomarker to predict prognosis in breast cancer and its bioinformatics analysis.World J Surg Oncol2026Its Methods give the RRID and name the linePubMed
HER2 Heterogeneous Breast Cancer Models Reveal Novel Therapeutic Targets and Subclonal Dynamics during Evolution to Resistance to HER2-Targeted Therapies.Cancer Discov2026Its Methods give the RRID and name the linePubMed
Integrated Multiomic Profiling Identifies BRD8/EP400 as a Pivotal Chromatin Module Mediating Anti-HER2 Response in HR+/HER2+ Breast Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Discovery of novel circular RNAs of the apoptosis-related BAX gene in breast cancer, by combining nanopore and next-generation sequencing.Mol Genet Genomics2026Its Methods give the RRID and name the linePubMed
Targeting TNBC: core-shell polycationic polyurea dendrimers with inherent anticancer activity.FEBS Open Bio2026Its Methods give the RRID and name the linePubMed
Combinatorial inhibition of IGF2BP3-regulated receptor tyrosine kinases offers a new therapeutic strategy for triple-negative breast cancer.J Exp Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Junction opener enables CAR T cell treatment of solid tumors.Sci Rep2026Its Methods give the RRID and name the linePubMed
Spermine Oxidase Serves as a Key Functional Node in Microbial Dysbiosis-Induced Breast Carcinogenesis.Cancer Res2026Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

For software

Read this list from software.

Twenty rows a page, as JSON. No key.

GET https://conductscience.org/api/cell-lines/v1/CVCL_0179/papers