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Cell line · CVCL_1161

Papers21

PaperJournalYearHow we knowRetractedSource
A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma.Cancer Res2026Its Methods give the RRID and name the linePubMed
Transcriptional regulation of protein synthesis by mediator kinase represents a therapeutic vulnerability in MYC-driven medulloblastoma.Nat Commun2025Its Methods give the RRID and name the linePubMed
Single-cell multi-omics identifies metabolism-linked epigenetic reprogramming as a driver of therapy-resistant medulloblastoma.Nat Commun2025Its Methods give the RRID and name the linePubMed
Metastatic medulloblastoma remodels the local leptomeningeal microenvironment to promote further metastatic colonization and growth.Nat Cell Biol2025Its Methods give the RRID and name the linePubMed
Single-cell multi-omics analysis identifies metabolism-linked epigenetic reprogramming as a driver of therapy-resistant medulloblastomaRes Sq2024Its Methods give the RRID and name the linePubMed
Transcriptional Regulation of Protein Synthesis by Mediator Kinase Represents a Therapeutic Vulnerability in MYC-driven MedulloblastomaRes Sq2024Its Methods give the RRID and name the linePubMed
Translation of non-canonical open reading frames as a cancer cell survival mechanism in childhood medulloblastoma.Mol Cell2024Its Methods give the RRID and name the linePubMed
Integrin-αvβ3 is a Therapeutically Targetable Fundamental Factor in Medulloblastoma Tumorigenicity and Radioresistance.Cancer Res Commun2023Its Methods give the RRID and name the linePubMed
Translation of non-canonical open reading frames as a cancer cell survival mechanism in childhood medulloblastoma.bioRxiv2023Its Methods give the RRID and name the linePubMed
Antiangiogenic Compound Axitinib Demonstrates Low Toxicity and Antitumoral Effects against Medulloblastoma.Cancers (Basel)2021Its Methods give the RRID and name the linePubMed
Transcriptional control of DNA repair networks by CDK7 regulates sensitivity to radiation in MYC-driven medulloblastoma.Cell Rep2021Its Methods give the RRID and name the linePubMed
Role of MYC-miR-29-B7-H3 in Medulloblastoma Growth and Angiogenesis.J Clin Med2019Its Methods give the RRID and name the linePubMed
Data on the number and frequency of scientific literature citations for established medulloblastoma cell lines.Data Brief2016Cellosaurus cites itPubMed
In vitro models of medulloblastoma: Choosing the right tool for the job.J Biotechnol2016Cellosaurus cites itPubMed
BET bromodomain inhibition of MYC-amplified medulloblastoma.Clin Cancer Res2014Cellosaurus cites itPubMed
MDM2 antagonism by nutlin-3 induces death in human medulloblastoma cells.Neurosci Lett2012Cellosaurus cites itPubMed
Neuralized1 causes apoptosis and downregulates Notch target genes in medulloblastoma.Neuro Oncol2010Cellosaurus cites itPubMed
Signatures of mutation and selection in the cancer genome.Nature2010Cellosaurus cites itPubMed
Chromosomal characteristics of childhood brain tumors.Cancer Genet Cytogenet1997Cellosaurus cites itPubMed
Differentiation characteristics of newly established medulloblastoma cell lines (D384 Med, D425 Med, and D458 Med) and their transplantable xenografts.Lab Invest1991Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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