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Gene · 10657

Papers1,948

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

TitleJournalYearPMIDFound bySource
CircLAMA5 traps Sam68 in the cytoplasm to regulate alternative splicing and drive sunitinib resistance in ccRCC.Oncogene202642744974PubTator, text miningPubMed
RAD51AP1-MYC-Sam68 positive transcriptional feedback loop maintains lung cancer stemness.iScience202642729436PubTator, text miningPubMed
MicroRNA-345-3p expression inhibits colorectal cancer liver metastasis by targeting KHDRBS1 and TGF-β signaling pathway.Hereditas202642723084PubTator, text miningPubMed
Sam68 stimulates polymerase theta-mediated end-joining by suppressing poison exon inclusion in Polq mRNA.Nucleic Acids Res202642578367PubTator, text miningPubMed
Multi-layer network biology combined with single-cell evidence reveals core regulatory nodes in atherosclerotic plaques and potential drug-targeting directions.Naunyn Schmiedebergs Arch Pharmacol202642410224PubTator, text miningPubMed
A comprehensive long RNA landscape of multi-regional porcine lung-derived small extracellular vesicles.BMC Genomics202642393508PubTator, text miningPubMed
Retraction notice to 'Sam68 impedes the recovery of arterial injury by augmenting inflammatory response' [Journal of Molecular and Cellular Cardiology 137 (2019) 82-92].J Mol Cell Cardiol202642303533PubTator, text miningPubMed
SAM68 is a multifunctional post-transcriptional regulator of cardiomyocyte differentiation.Nucleic Acids Res202642240622PubTator, text miningPubMed
ENOX2 and the related mechanisms as potential genetic targets for varicose veins of lower extremities: An integrated bioinformatics and Mendelian analysis.Medicine (Baltimore)202642216315PubTator, text miningPubMed
Beyond SMN1 mutation: environmental predispositions and epigenetic contributors to phenotypic variability and disease severity in spinal muscular atrophy.Neuroscience202642105903PubTator, text miningPubMed
Retinoic Acid Receptor Responder Protein 1 Promotes Tubular Fibrosis via the KH RNA Binding Domain Containing, Signal Transduction Associated 1/Steroid Receptor Coactivator/p-Signal Transducer and Activator of Transcription 3 Axis.
J Am Soc Nephrol202641955023PubTator, text miningPubMed
High-content CRISPR activation screens identify synthetically lethal RNA-based mechanisms to sensitize cancer cells to targeted T cell cytotoxicity.Nat Genet202641946842PubTator, text miningPubMed
Stress granules at the crossroads of retroviral replication and antiviral immunity: mechanisms and therapeutic opportunities.Mol Biol Rep202641931190PubTator, text miningPubMed
Correction to 'Sam68 functions as a transcriptional coactivator of the p53 tumor suppressor'.Nucleic Acids Res202641841498PubTator, text miningPubMed
RNA binding protein Sam68 promotes germinal center reaction and IgG response through regulation of miR29.Cell Mol Life Sci202641792467PubTator, text miningPubMed
U2AF2 controls alternative splicing in speckle-proximal regions in an RS domain-dependent manner.Nucleic Acids Res202641761909PubTator, text miningPubMed
Emerging Protein Targets in Triple-Negative Breast Cancer: Beyond Conventional Therapy.Cancers (Basel)202641749871PubTator, text miningPubMed
Human papillomavirus 16 E6 oncoprotein promotes up-regulation of RNA-binding protein Sam68 in head and neck cancer.Front Microbiol202641725810PubTator, text miningPubMed
Integrated multi-omics analysis of prognostic model and immune microenvironment in intrahepatic cholangiocarcinoma.Transl Oncol202641558145PubTator, text miningPubMed
[Retracted] Clinical significance and effect of Sam68 expression in gastric cancer.Oncol Lett202641393073PubTator, text miningPubMed

gene-search-2026-11-11-0.2.0

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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