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Gene · 2053

Papers2,484

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

TitleJournalYearPMIDFound bySource
Distinct lipid mediator profiles in brain and liver tissues in Alzheimer's disease.medRxiv202642818430PubTator, text miningPubMed
Network toxicology and machine learning analysis of MC-LR-associated hepatocellular carcinoma.J Environ Sci Health B202642811799PubTator, text miningPubMed
Soluble epoxide hydrolase research, international collaboration, and translational capacity in Brazil: the scientific legacy of Bruce D. Hammock.Drug Metab Dispos202642805148PubTator, text miningPubMed
Anti-Inflammatory Activity of Monoterpene Phenol Dimers from Satureja bachtiarica.ACS Omega202642799005PubTator, text miningPubMed
EPHX2 Loss Promotes CRC Migration via EET-Dependent Chemokine Elevation and Mitochondrial Defect.Cancer Sci202642732782PubTator, text miningPubMed
Altered Epoxyeicosatrienoic Acid Signaling in Advanced-Stage Alzheimer's Disease (Investigation of Epoxyeicosatrienoic Acid Levels in Patients with Advanced Alzheimer's Disease).Curr Alzheimer Res202642725525PubTator, text miningPubMed
The Roles of EETs in Neurological Disorders.CNS Neurosci Ther202642711888PubTator, text miningPubMed
Design, synthesis, and biological evaluation of novel pyrimidine-sulfonamide derivatives as multi-targeted COX-2/5-LOX/sEH inhibitors with potential antiproliferative properties.RSC Adv202642662511PubTator, text miningPubMed
EPHX2 Expression and Its Association with Prognosis, Metabolic Regulation, and Metastasis-Related Pathways in Lung Adenocarcinoma.Genes (Basel)202642650154PubTator, text miningPubMed
Retraction notice to "Reducing degradation of EETs by sEH protein macromolecule depletion prevents MCAO and inhibits microglia-mediated inflammatory response" [Int. J. Biol. Macromol. 317 (2025) 144820].
Int J Biol Macromol202642613210PubTator, text miningPubMed
Topliss-Guided Optimization of Acetanilide-Based Soluble Epoxide Hydrolase Inhibitors: Exploring the Pentafluorosulfanyl Group as a Potency-Enhancing Motif.ChemMedChem202642596002PubTator, text miningPubMed
Alterations in Epoxyeicosatrienoic Acids Metabolism in Type 2 Diabetes Mellitus and Potential Association with Retinopathy Progression.Curr Med Chem202642487591PubTator, text miningPubMed
Modular Access to Diverse Soluble Epoxide Hydrolase Degraders as Potential Agents for Alleviating LPS-Induced Acute Inflammation.J Med Chem202642469598PubTator, text miningPubMed
Placental abundance of arachidonic acid metabolites is reduced in preterm placentas but is not altered in fetal growth restriction.Prostaglandins Leukot Essent Fatty Acids202642447711PubTator, text miningPubMed
Bioinformatics analysis reveals the association of bile acid metabolism-related genes with sepsis.PLoS One202642430416PubTator, text miningPubMed
Identification of soluble epoxide hydrolase inhibitors from natural products via structure-based virtual screening, in vitro assays, and molecular dynamics simulations.Mol Divers202642423921PubTator, text miningPubMed
Integration of lung tissue proteomics and genome-wide association data to identify lung cancer susceptibility proteins and potential drug targets.medRxiv202642396338PubTator, text miningPubMed
Shared genetic architecture of schizophrenia and Alzheimer's disease and related dementias implicates 16p11.2 and lifespan brain vulnerability.Mol Psychiatry202642387103PubTator, text miningPubMed
Steroidogenesis suppression in H295R cells by 1,3-disubstituted ureas: A potential off-target effect of some sEH inhibitors.Toxicol Lett202642365935PubTator, text miningPubMed
Design, Synthesis, and Biological Evaluation of Tetrahydroindazole-Based Sulfonamides as Potential Multi-Target Anti-Inflammatory Agents.Pharmaceuticals (Basel)202642356461PubTator, text miningPubMed

gene-search-2026-11-11-0.2.0

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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