Gene · 2230
Papers1,417
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
121 to 140 of 1,417
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| GCSH promotes colorectal cancer progression by inhibiting Cuproptosis through the PI3K/AKT-FDX1 axis. | Funct Integr Genomics | 2026 | 41591502 | PubTator, text mining | PubMed |
| Beyond metabolism: exploring the regulatory and therapeutic implications of lactate and lactylation in cancer-regulated cell death. | Cell Death Dis | 2026 | 41571623 | PubTator, text mining | PubMed |
| Metal-based nanomaterials achieve tumor treatment strategies by overcoming the blood-brain barrier of glioma and responding to the tumor microenvironment. | Mater Today Bio | 2026 | 41560814 | PubTator, text mining | PubMed |
| Ginsenoside Ro alleviated cuproptosis induced by high oxalate via inhibiting zinc transporter ZnT1 in renal tubular epithelial cells. | J Ginseng Res | 2026 | 41550586 | PubTator, text mining | PubMed |
| Cuproptosis of bronchial epithelial cells triggers airway remodeling in chronic asthma. | Sci Rep | 2026 | 41530300 | PubTator, text mining | PubMed |
| FDX1-mediated cuproptosis promotes cholestatic liver injury exacerbated by taurocholic acid-enhanced copper accumulation. | Cell Death Discov | 2026 | 41513631 | PubTator, text mining | PubMed |
| METTL3-mediated m6A modification of FDX1 confers resistance to cuproptosis and promotes hepatocellular carcinoma progression. | Commun Biol | 2026 | 41501140 | PubTator, text mining | PubMed |
| Mitochondrial uncoupling sensitizes gastric cancer cells to elesclomol-induced cuproptosis via FDX1/DLAT upregulation. | Free Radic Biol Med | 2026 | 41407052 | PubTator, text mining | PubMed |
| Dual-functional copper nanoplatform potentiates cuproptosis through p53 reactivation and metabolic reprogramming. | J Colloid Interface Sci | 2026 | 41352051 | PubTator, text mining | PubMed |
| A biodegradable nanomedicine for potentiated cancer therapy via Ca2+-overload and photothermal dual-amplifying cuproptosis. | J Colloid Interface Sci | 2026 | 41308482 | PubTator, text mining | PubMed |
| Recent advances in the study of cuproptosis in gliomas. | Int Immunopharmacol | 2026 | 41273844 | PubTator, text mining | PubMed |
| Copper-induced duodenal injury: Unveiling the dual role of cuproptosis and ferroptosis via the FDX1/GPX4 axis. | J Nutr Biochem | 2026 | 41224149 | PubTator, text mining | PubMed |
| Cigarette smoke exposure diminishes ovarian reserve in mice by regulating granulosa cells redox homeostasis imbalance through Wnt10b-ERβ feedback loop. | Chem Biol Interact | 2026 | 41202950 | PubTator, text mining | PubMed |
| Fe-S Protein FDX1 Triggers Tumor-Intrinsic Innate Immunity via Mitochondrial Nucleic Acids Release to Orchestrate Ferroptosis in CCRCC. | Adv Sci (Weinh) | 2026 | 41199656 | PubTator, text mining | PubMed |
| SIRT7 deficiency promoted cuproptosis-mediated mitochondrial dysfunction and inhibited malignant development of cervical cancer. | Arch Biochem Biophys | 2026 | 41167446 | PubTator, text mining | PubMed |
| The CEBPA/FDX1 axis elevates sensitivity to cuproptosis in lung adenocarcinoma cells. | Biochim Biophys Acta Gen Subj | 2026 | 41135638 | PubTator, text mining | PubMed |
| LIAS Promotes Cuproptosis in Prostate Cancer Cells by Suppressing Glycolysis via the p53 Signaling Pathway. | Prostate | 2026 | 41031841 | PubTator, text mining | PubMed |
| The pathogenesis of liver fibrosis in Wilson's disease: hepatocyte injury and regulation mediated by copper metabolism dysregulation. | Biometals | 2026 | 41006805 | PubTator, text mining | PubMed |
| Clinical significance and immune microenvironment association of cuproptosis-related genes in pan-cancer. | Exp Physiol | 2026 | 40560740 | PubTator, text mining | PubMed |
| Associations between carotid artery intima-media thickness, traditional risk factors and proteins. | NPJ Cardiovasc Health | 2025 | 41775896 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this list from software.
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GET https://conductscience.org/api/genes/v1/2230/papers?page=7