Gene · 3776
Papers1,439
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
81 to 100 of 1,439
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| Development of covalent chemogenetic K2P channel activators. | Cell Chem Biol | 2024 | 39029456 | PubTator, text mining | PubMed |
| Pantoprazole and riluzole target H+/K+-ATPases and pH-sensitive K+ channels in pancreatic cancer cells. | Int J Cancer | 2024 | 38975879 | PubTator, text mining | PubMed |
| Activation of hTREK-1 by polyunsaturated fatty acids involves direct interaction. | Sci Rep | 2024 | 38956407 | PubTator, text mining | PubMed |
| Electrophysiological Effects of the Sodium-Glucose Co-Transporter-2 (SGLT2) Inhibitor Dapagliflozin on Human Cardiac Potassium Channels. | Int J Mol Sci | 2024 | 38891889 | PubTator, text mining | PubMed |
| Piezo1 ion channels are capable of conformational signaling. | bioRxiv | 2024 | 38854150 | PubTator, text mining | PubMed |
| The TREK-1 potassium channel is involved in both the analgesic and anti-proliferative effects of riluzole in bone cancer pain. | Biomed Pharmacother | 2024 | 38852511 | PubTator, text mining | PubMed |
| TREK2 Lipid Binding Preferences Revealed by Native Mass Spectrometry. | J Am Soc Mass Spectrom | 2024 | 38843438 | PubTator, text mining | PubMed |
| Atomistic mechanism of coupling between cytosolic sensor domain and selectivity filter in TREK K2P channels. | Nat Commun | 2024 | 38821927 | PubTator, text mining | PubMed |
| The TREK-1 potassium channel is a potential pharmacological target for vasorelaxation in pulmonary hypertension. | Br J Pharmacol | 2024 | 38807478 | PubTator, text mining | PubMed |
| Extracellular modulation of TREK-2 activity with nanobodies provides insight into the mechanisms of K2P channel regulation. | Nat Commun | 2024 | 38755204 | PubTator, text mining | PubMed |
| The Inhibition of TREK-1 K+ Channels via Multiple Compounds Contained in the Six Kamikihito Components, Potentially Stimulating Oxytocin Neuron Pathways. | Int J Mol Sci | 2024 | 38732124 | PubTator, text mining | PubMed |
| The Variety of Mechanosensitive Ion Channels in Retinal Neurons. | Int J Mol Sci | 2024 | 38732096 | PubTator, text mining | PubMed |
| Sortilin-Mediated Inhibition of TREK1/2 Channels in Primary Sensory Neurons Promotes Prediabetic Neuropathic Pain. | Adv Sci (Weinh) | 2024 | 38626370 | PubTator, text mining | PubMed |
| Tension activation of mechanosensitive two-pore domain K+ channels TRAAK, TREK-1, and TREK-2. | Nat Commun | 2024 | 38605031 | Swiss-Prot, curated | PubMed |
| Long-term follow-up of TREK-1 KO mice reveals the development of bladder hypertrophy and impaired bladder smooth muscle contractility with age. | Am J Physiol Renal Physiol | 2024 | 38601986 | PubTator, text mining | PubMed |
| Up-regulated expression of two-pore domain K+ channels, KCNK1 and KCNK2, is involved in the proliferation and migration of pulmonary arterial smooth muscle cells in pulmonary arterial hypertension. | Front Cardiovasc Med | 2024 | 38410243 | PubTator, text mining | PubMed |
| Mechanical activation of TWIK-related potassium channel by nanoscopic movement and rapid second messenger signaling. | Elife | 2024 | 38407149 | PubTator, text mining | PubMed |
| Assessment of Research Ethics Knowledge of Pediatricians. | Glob Pediatr Health | 2024 | 38269316 | PubTator, text mining | PubMed |
| Enhancing Human Cutaneous Wound Healing through Targeted Suppression of Large Conductance Ca2+-Activated K+ Channels. | Int J Mol Sci | 2024 | 38255877 | PubTator, text mining | PubMed |
| KCNK5 Regulating Potassium Efflux and Inducing Pyroptosis in Corneal Epithelial Cells Through TNFSF10-Mediated Autophagy in Dry Eye. | Invest Ophthalmol Vis Sci | 2024 | 38236186 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this list from software.
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GET https://conductscience.org/api/genes/v1/3776/papers?page=5