ConductScience.org, home
YTHDF1: YTH N6-methyladenosine RNA binding protein F1
API referenceFor AI assistants

Gene · 54915

Papers3,036

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

1 to 20 of 3,036

Download this page as JSON
TitleJournalYearPMIDFound bySource
Single residues within the YTHDF intrinsically disordered region control m 6 A-dependent mRNA decay.bioRxiv202642818126PubTator, text miningPubMed
Epitranscriptomic regulation of Notch signaling.Cell Signal202642800623PubTator, text miningPubMed
METTL3‑mediated OTUB1 sustains PD‑L1 protein stability via deubiquitination and promotes immune evasion in oral squamous cell carcinoma.Mol Cell Biochem202642776380PubTator, text miningPubMed
Lactylation and N6-Methyladenosine RNA modification in neuropsychiatric sequelae after intracerebral hemorrhage: a hypothesis-generating metabolic-glial-circuit framework.Mol Biol Rep202642730969PubTator, text miningPubMed
NBR1-Mediated Autophagic Degradation of YTHDF1 Curtails FDX1 Translation to Drive Concurrent Multikinase Inhibitor Resistance and Cuproptosis Tolerance.Cancer Commun (Lond)202642729649PubTator, text miningPubMed
Mutant KRAS-m6A Epitranscriptome Axis Promotes Colorectal Cancer and is a Therapeutic Target.Adv Sci (Weinh)202642717493PubTator, text miningPubMed
Development of m6A Readers for Enhanced Recognition In Vitro via Genetic Code Expansion.ACS Chem Biol202642715419PubTator, text miningPubMed
RNA m⁶A modification and its regulatory factors are altered in granulosa cells from women with PMOS.J Assist Reprod Genet202642687053PubTator, text miningPubMed
YTHDF1-mediated m6A RNA regulation controls cardiomyocyte remodeling through KLF11-HIF1α.Proc Natl Acad Sci U S A202642679039PubTator, text miningPubMed
Single-cell RNA sequencing uncovers m6A regulator-defined T-cell subpopulations and YTHDF1-HSP90AB1-MYC therapeutic axis in T-cell acute lymphoblastic leukemia.Chin Med J (Engl)202642675534PubTator, text miningPubMed
Role and prognostic value of N6-methyladenosine RNA modification regulators in glycolytic reprogramming during sepsis.J Intensive Care202642675521PubTator, text miningPubMed
METTL3-mediated m6A modification exacerbates vascular endothelial inflammation in coronary artery disease by regulating DNMT1 mRNA stability.Exp Cell Res202642674138PubTator, text miningPubMed
Gene Variations in RNA Modification Pathway Linked to Poor Survival After Gastric Cancer Surgery.Genes (Basel)202642650134PubTator, text miningPubMed
YTHDF1-mediated translational activation of SETD2 suppresses aerobic glycolysis and sensitizes renal cell carcinoma to sunitinib.Cell Signal202642648585PubTator, text miningPubMed
Comprehensive Analysis of m6A Regulators Identifies YTHDF3 as a Promising Prognostic Biomarker for Breast Cancer.J Vis Exp202642612048PubTator, text miningPubMed
Ythdf family m6A readers promote retinal ganglion cell fate reprogramming and neurite development.Exp Eye Res202642595204PubTator, text miningPubMed
Extracellular Vesicles in Cardiovascular Disease: Intercellular Signaling, Liquid Biopsy Biomarkers, and Therapeutic Translation.Circ Res202642594169PubTator, text miningPubMed
METTL4 deficiency alleviates sepsis-induced liver injury via inhibiting NCOA4-mediated ferritinophagy and ferroptosis.Cell Signal202642571828PubTator, text miningPubMed
MARVELD3 as a Prognostic Biomarker in Esophageal Cancer and its Relationship with m6A Modification, Cuproptosis, and the ceRNA Regulatory Network.Curr Gene Ther202642517389PubTator, text miningPubMed
METTL3-YTHDF1-driven m6A enhancement of MYC signaling orchestrates DNA repair and metabolic reprogramming to promote chemoresistance in colorectal cancer.Mol Cell Biochem202642418117PubTator, text miningPubMed

gene-search-2026-11-11-0.2.0

Next page

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this list from software.

Twenty rows a page, as JSON. No key.

GET https://conductscience.org/api/genes/v1/54915/papers

API referenceFor AI assistants