Gene · 54915
Papers3,036
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
221 to 240 of 3,036
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| SETD5 facilitates stemness and represses ferroptosis via m6A-mediating PKM2 stabilization in non-small cell lung cancer. | Oncogene | 2025 | 40307507 | PubTator, text mining | PubMed |
| The METTL14-YTHDF1-SAP30 Axis Promotes Glycolysis and Oxaliplatin Resistance in Colorectal Adenocarcinoma via m6A Modification. | J Gastroenterol Hepatol | 2025 | 40289460 | PubTator, text mining | PubMed |
| YTHDF1-mediated m6A modification promotes cisplatin resistance in ovarian cancer via the FZD7/Wnt/β-catenin pathway. | Apoptosis | 2025 | 40281310 | PubTator, text mining | PubMed |
| Effects of Benzo[a]Pyrene Exposure on Lung Cancer: A Mechanistic Study of Epigenetic m6A Levels and YTHDF1. | Toxics | 2025 | 40278596 | PubTator, text mining | PubMed |
| YTHDF1/RNF7/p27 axis promotes prostate cancer progression. | Cell Death Dis | 2025 | 40251202 | PubTator, text mining | PubMed |
| Epigenetics of maternal-fetal interface immune microenvironment and placental related pregnancy complications. | Front Immunol | 2025 | 40248704 | PubTator, text mining | PubMed |
| Nuclear accumulation of YTHDF1 regulates mRNA splicing in the DNA damage response. | Sci Adv | 2025 | 40238889 | PubTator, text mining | PubMed |
| YTHDF1 promotes pancreatic cancer cell progression by enhancing SF3B2 translation though m6A modification. | J Biochem | 2025 | 40211730 | PubTator, text mining | PubMed |
| m6A demethylase ALKBH5 reduces ferroptosis in diabetic retinopathy through the m6A-YTHDF1-ACSL4 axis. | Diabet Med | 2025 | 40210448 | PubTator, text mining | PubMed |
| FOXD2-AS1 is modulated by METTL3 with the assistance of YTHDF1 to affect proliferation and apoptosis in esophageal cancer. | Acta Pharm | 2025 | 40208783 | PubTator, text mining | PubMed |
| Demethylase FTO mediates m6A modification of ENST00000619282 to promote apoptosis escape in rheumatoid arthritis and the intervention effect of Xinfeng Capsule. | Front Immunol | 2025 | 40181982 | PubTator, text mining | PubMed |
| The inducible role of autophagy in cell death: emerging evidence and future perspectives. | Cell Commun Signal | 2025 | 40140912 | PubTator, text mining | PubMed |
| m6A-induced DEAD-box RNA helicase 21 enhances lipid metabolism via 3‑hydroxy-3-methylglutaryl-CoA synthases 1 in colorectal cancer. | Transl Oncol | 2025 | 40127603 | PubTator, text mining | PubMed |
| Coupling mechanisms coordinating mRNA translation with stages of the mRNA lifecycle. | RNA Biol | 2025 | 40116043 | PubTator, text mining | PubMed |
| Single-cell sequencing analysis reveals the essential role of the m 6A reader YTHDF1 in retinal visual function by regulating TULP1 and DHX38 translation. | Zool Res | 2025 | 40116022 | PubTator, text mining | PubMed |
| M6A-modified BFSP1 induces aerobic glycolysis to promote liver cancer growth and metastasis through upregulating tropomodulin 4. | Mol Biomed | 2025 | 40097750 | PubTator, text mining | PubMed |
| METTL3-m6A-mediated TGF-β signaling promotes Fuchs endothelial corneal dystrophy via regulating corneal endothelial-to-mesenchymal transition. | Cell Death Discov | 2025 | 40089501 | PubTator, text mining | PubMed |
| Specific sDMA modifications on the RGG/RG motif of METTL14 regulate its function in AML. | Cell Commun Signal | 2025 | 40057764 | PubTator, text mining | PubMed |
| SLC31A1 loss depletes mitochondrial copper and promotes cardiac fibrosis. | Eur Heart J | 2025 | 40048660 | PubTator, text mining | PubMed |
| METTL3/YTDHF1 Stabilizes MTCH2 mRNA to Regulate Ferroptosis in Glioma Cells. | Front Biosci (Landmark Ed) | 2025 | 40018930 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
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GET https://conductscience.org/api/genes/v1/54915/papers?page=12