Gene · 57192
Papers1,559
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
101 to 120 of 1,559
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| Acid Sphingomyelinase Regulates AdipoRon-Induced Differentiation of Arterial Smooth Muscle Cells via TFEB Activation. | Int J Mol Sci | 2025 | 40076784 | PubTator, text mining | PubMed |
| PFOS causes lysosomes-regulated mitochondrial fission through TRPML1-VDAC1 and oligomerization of MCU/ATP5J2. | J Hazard Mater | 2025 | 39983639 | PubTator, text mining | PubMed |
| Lysosomal Fe2+ influx through MCOLN1 channel prevents sustained inflammation by limiting PHDs-regulated NFKB activation in macrophages. | Autophagy | 2025 | 39936589 | PubTator, text mining | PubMed |
| Pharmacological activation of TRPML1 enhances autophagy regulating hypertonicity and TGF-β-induced EMT in proximal tubular epithelial cells. | Biochem Biophys Res Commun | 2025 | 39893888 | PubTator, text mining | PubMed |
| Lysosomes finely control macrophage inflammatory function via regulating the release of lysosomal Fe2+ through TRPML1 channel. | Nat Commun | 2025 | 39856099 | PubTator, text mining | PubMed |
| Corrigendum to "Jingfang Granule mitigates Coxsackievirus B3-induced myocardial damage by modulating mucolipin 1 expression" [J. Ethnopharmacol. 320 (2024) 117396]. | J Ethnopharmacol | 2025 | 39613537 | PubTator, text mining | PubMed |
| Activation of lysosomal Ca2+ channels mitigates mitochondrial damage and oxidative stress. | J Cell Biol | 2025 | 39500490 | PubTator, text mining | PubMed |
| Lysosomal cation channel TRPML1 suppression sensitizes acute myeloid leukemia cells to chemotherapeutics by inhibiting autophagy. | Mol Cell Biochem | 2025 | 38951379 | PubTator, text mining | PubMed |
| Multimodal Imaging in Mucolipidosis Type IV: Siblings With Novel Genetic Variant. | Cornea | 2025 | 38913974 | PubTator, text mining | PubMed |
| Lysosomal Channels as New Molecular Targets in the Pharmacological Therapy of Neurodegenerative Diseases via Autophagy Regulation. | Curr Neuropharmacol | 2025 | 38766825 | PubTator, text mining | PubMed |
| A Novel Homozygous Variant in the MCOLN1 Gene Associated With Severe Oromandibular Dystonia and Parkinsonism. | Can J Neurol Sci | 2025 | 38532569 | PubTator, text mining | PubMed |
| Getting PIKy with the lysosome membrane (again). | Autophagy Rep | 2024 | 40395513 | PubTator, text mining | PubMed |
| Editorial: 73rd annual meeting of the Italian society of physiology: advancement in basic and translational physiology. | Front Physiol | 2024 | 39839533 | PubTator, text mining | PubMed |
| LAMTOR1 regulates dendritic lysosomal positioning in hippocampal neurons through TRPML1 inhibition. | Front Cell Neurosci | 2024 | 39650798 | PubTator, text mining | PubMed |
| TRPML1 agonist ML-SA5 mitigates uranium-induced nephrotoxicity via promoting lysosomal exocytosis. | Biomed Pharmacother | 2024 | 39647321 | PubTator, text mining | PubMed |
| TRPML1 acts as a predisposing factor in lymphedema development by regulating the subcellular localization of aquaporin-3, -5. | PLoS One | 2024 | 39637010 | PubTator, text mining | PubMed |
| Nanoparticles encapsulating phosphatidylinositol derivatives promote neuroprotection and functional improvement in preclinical models of ALS via a long-lasting activation of TRPML1 lysosomal channel. | Pharmacol Res | 2024 | 39491634 | PubTator, text mining | PubMed |
| Lysosomal Ca2+ release-facilitated TFEB nuclear translocation alleviates ischemic brain injury by attenuating autophagic/lysosomal dysfunction in neurons. | Sci Rep | 2024 | 39438678 | PubTator, text mining | PubMed |
| Lysosomal activity depends on TRPML1-mediated Ca2+ release coupled to incoming vesicle fusions. | J Biol Chem | 2024 | 39433126 | PubTator, text mining | PubMed |
| TRPML1 gating modulation by allosteric mutations and lipids. | Elife | 2024 | 39400550 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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