Gene · 5867
Papers1,548
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
21 to 40 of 1,548
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| CTLA4-related primary immune dysregulatory disorders. | Curr Opin Allergy Clin Immunol | 2025 | 41158012 | PubTator, text mining | PubMed |
| Extracellular vesicles: cargo loading, degradation and secretory pathways, and their intersection with autophagy. | Extracell Vesicles Circ Nucl Acids | 2025 | 41132500 | PubTator, text mining | PubMed |
| Rab10 inactivation promotes AMPAR trafficking and spine enlargement during long-term potentiation. | Elife | 2025 | 40986440 | PubTator, text mining | PubMed |
| Nanoparticle-Delivered siRNA Targeting NSUN4 Relieves Systemic Lupus Erythematosus through Declining Mitophagy-Mediated CD8+T Cell Exhaustion. | MedComm (2020) | 2025 | 40761479 | PubTator, text mining | PubMed |
| TRPML2 channel modulation by PI(3,5)P₂ and small-molecule agonists controls endosomal vesicle dynamics. | Biomed Pharmacother | 2025 | 40664038 | PubTator, text mining | PubMed |
| Emerging technologies towards extracellular vesicles large-scale production. | Bioact Mater | 2025 | 40585384 | PubTator, text mining | PubMed |
| Rab10 inactivation promotes AMPAR trafficking and spine enlargement during long-term potentiation. | bioRxiv | 2025 | 40501544 | PubTator, text mining | PubMed |
| Loss-of-function Gαs rare disease variants exert mutation-specific effects on GPCR signaling. | Sci Signal | 2025 | 40392940 | PubTator, text mining | PubMed |
| Induced mitochondrial deficit by NDUFS3 transient silencing reduces RAB7 expression and causes lysosomal dysfunction in pancreatic cancer cells. | Cell Commun Signal | 2025 | 40369571 | PubTator, text mining | PubMed |
| Interphase cell morphology defines the mode, symmetry, and outcome of mitosis. | Science | 2025 | 40310923 | PubTator, text mining | PubMed |
| The mechanisms of HER2 targeted ADCs are dependent on Rab GTPases. | Ther Adv Med Oncol | 2025 | 40297622 | PubTator, text mining | PubMed |
| A new Similarity Based Adapted Louvain Algorithm (SIMBA) for active module identification in p-value attributed biological networks. | Sci Rep | 2025 | 40175439 | PubTator, text mining | PubMed |
| Nucleolin in the cell membrane promotes Ang II-mediated VSMC phenotypic switching by regulating the AT1R internalization function : Nucleolin promotes Ang II-mediated VSMC phenotypic switching. | Biol Direct | 2025 | 40001211 | PubTator, text mining | PubMed |
| PI3KC2β depletion rescues endosomal trafficking defects in Mtm1 knockout skeletal muscle cells. | J Lipid Res | 2025 | 39952567 | PubTator, text mining | PubMed |
| Rab7a is required to degrade select blood-brain barrier junctional proteins after ischemic stroke. | bioRxiv | 2025 | 37693406 | PubTator, text mining | PubMed |
| BEACH domain proteins in membrane trafficking and disease. | J Cell Biol | 2024 | 39526996 | PubTator, text mining | PubMed |
| Deciphering the role of lipid metabolism-related genes in Alzheimer's disease: a machine learning approach integrating Traditional Chinese Medicine. | Front Endocrinol (Lausanne) | 2024 | 39507054 | PubTator, text mining | PubMed |
| RAB4A is a master regulator of cancer cell stemness upstream of NUMB-NOTCH signaling. | Cell Death Dis | 2024 | 39463384 | PubTator, text mining | PubMed |
| -RAMP3 promotes hepatocellular carcinoma tumor cell-mediated CCL2 degradation by supporting membrane distribution of ACKR2. | Int Immunopharmacol | 2024 | 39437486 | PubTator, text mining | PubMed |
| Arf1-dependent LRBA recruitment to Rab4 endosomes is required for endolysosome homeostasis. | J Cell Biol | 2024 | 39325073 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this list from software.
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GET https://conductscience.org/api/genes/v1/5867/papers?page=2