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Gene · 779

Papers2,128

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

1,101 to 1,120 of 2,128

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TitleJournalYearPMIDFound bySource
Review of the Diagnosis and Treatment of Periodic Paralysis.Muscle Nerve201829125635PubTator, text miningPubMed
Diverse phenotype of hypokalaemic periodic paralysis within a family.Pract Neurol201828972032PubTator, text miningPubMed
Sodium Channelopathies of Skeletal Muscle.Handb Exp Pharmacol201828939973PubTator, text miningPubMed
ING5 activity in self-renewal of glioblastoma stem cells via calcium and follicle stimulating hormone pathways.Oncogene201828925404PubTator, text miningPubMed
Malignant Hyperthermia in the Post-Genomics Era: New Perspectives on an Old Concept.Anesthesiology201828902675PubTator, text miningPubMed
Treating pediatric neuromuscular disorders: The future is now.Am J Med Genet A201828889642PubTator, text miningPubMed
Relevance of tissue specific subunit expression in channelopathies.Neuropharmacology201828669898PubTator, text miningPubMed
Trafficking of neuronal calcium channels.Neuronal Signal201732714572PubTator, text miningPubMed
Structure-Function Relationship of the Voltage-Gated Calcium Channel Cav1.1 Complex.Adv Exp Med Biol201729594856PubTator, text miningPubMed
Genetic variation in human drug-related genes.Genome Med201729273096PubTator, text miningPubMed
De novo reconstitution reveals the proteins required for skeletal muscle voltage-induced Ca2+ release.Proc Natl Acad Sci U S A201729229815PubTator, text miningPubMed
Voltage-dependent calcium channel signaling mediates GABAA receptor-induced migratory activation of dendritic cells infected by Toxoplasma gondii.PLoS Pathog201729216332PubTator, text miningPubMed
Alternative Splicing of L-type CaV1.2 Calcium Channels: Implications in Cardiovascular Diseases.Genes (Basel)201729186814PubTator, text miningPubMed
Congenital myopathies: clinical phenotypes and new diagnostic tools.Ital J Pediatr201729141652PubTator, text miningPubMed
An atypical CaV1.1 mutation reveals a common mechanism for hypokalemic periodic paralysis.J Gen Physiol201729138267PubTator, text miningPubMed
The structural basis of ryanodine receptor ion channel function.J Gen Physiol201729122978PubTator, text miningPubMed
Na leak with gating pore properties in hypokalemic periodic paralysis V876E mutant muscle Ca channel.J Gen Physiol201729114033PubTator, text miningPubMed
Structural insights into binding of STAC proteins to voltage-gated calcium channels.Proc Natl Acad Sci U S A201729078335Swiss-Prot, curatedPubMed
Functional expression and pharmaceutical efficacy of cardiac-specific ion channels in human embryonic stem cell-derived cardiomyocytes.Sci Rep201729062050PubTator, text miningPubMed
THE CONCISE GUIDE TO PHARMACOLOGY 2017/18: Voltage-gated ion channels.Br J Pharmacol201729055033PubTator, text miningPubMed

gene-search-2026-11-11-0.2.0

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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