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Gene · 8438

RAD54L: RAD54 like

12 sourcesgene-search-2026-11-11-0.2.0

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ConductScience Library. RAD54L: RAD54 like. Genes, record 8438. Release gene-search-2026-11-11-0.2.0, 11 Nov 2026. https://conductscience.org/library/genes/8438

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Gene

Symbol
RAD54L
HGNC
Name
RAD54 like
HGNC
NCBI Gene ID
8438
NCBI Gene
HGNC ID
HGNC:9826
HGNC
Ensembl
ENSG00000085999
NCBI Gene
Aliases
HR54, RAD54A, hHR54, hRAD54
NCBI Gene
Type
protein-coding
NCBI Gene

Proteins1

Reviewed human proteins whose Swiss-Prot entry states the gene.

UniProt entryProteinLengthStructuresGene stated in
Q92698DNA repair and recombination protein RAD54-like7471Ensembl, GeneID, HGNC

Source UniProt

Patents33

Patents whose sequence listing contains this protein's exact sequence. A patent can list a sequence without claiming it.

PublicationPublishedSEQ ID NOProteinSource
EP 4641201 A22025-10-299305Q92698Espacenet
EP 4641202 A22025-10-295533Q92698Espacenet
JP 2025517969 A2025-06-129305Q92698Espacenet
KR 1020250017231 A2025-02-045533Q92698Espacenet
JP 2024123008 A2024-09-108045Q92698Espacenet
JP 2024123008 A2024-09-108046Q92698Espacenet
US 11976334 B22024-05-07196Q92698Espacenet
US 11446398 B22022-09-2071189Q92698Espacenet
US 11446398 B22022-09-2071190Q92698Espacenet
JP 2022092366 A2022-06-22803Q92698Espacenet

1 to 10 of 33

See all 33 patents

Drugs0

Drugs a ChEMBL mechanism record says act on the gene's protein, through Open Targets.

None in this release.

Trials0

Trials of those drugs. A trial joins the gene only through a drug that acts on it.

None in this release.

NIH grants1,244

NIH grants whose papers are about the gene, through NIH's grant-paper links.

GrantTitleRAD54L papersNewest fiscal year
R01GM058015Recombinational Mechanisms of DNA Repair262025
P01CA092584Structural Cell Biology of DNA Repair Machines222025
P30CA008748Cancer Center Support Grant192025
R01ES007061Mechanisms of DNA Double-strand Break End Resection and Repair Pathway Choice172025
P30CA016672Cancer Center Support Grant162025
R35GM118026Mechanisms of Genome Integrity112025
ZIAAG000741Roles Of DNA Helicases In Pathways Required For Maintenance Of Genomic Stability102025
P30CA006973Regional Oncology Research Center92025
P30CA056036Translational Research in Cancer82025
P30CA015704Cancer Center Support Grant72025

1 to 10 of 1,244Source NIH's project files

See all 1,244 nih grants

Papers1,865

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

TitleJournalYearPMIDFound bySource
MCPH1 and DNA Damage Response Pathway Genes as Candidate Susceptibility Loci in Hereditary Breast Cancer.Diagnostics (Basel)202642793752PubTator, text miningPubMed
Whole-genome sequencing identifies a potentially causative homozygous splicing variant in RAD54 like in non-obstructive azoospermia.Int J Reprod Biomed202642774555PubTator, text miningPubMed
The dynamics of RAD51 foci formation and elongation in living human cells.Nucleic Acids Res202642725434PubTator, text miningPubMed
RAD54L coordinates the nucleolar DNA damage response to maintain rDNA stability.EMBO J202642493648PubTator, text miningPubMed
Role of chromatin remodeling and immunopharmacology in cancer therapy.Mol Med202642458295PubTator, text miningPubMed
Real-world clinical outcomes, genetic testing patterns, and treatment patterns of patients with metastatic castration-resistant prostate cancer treated with olaparib monotherapy in the United States.Urol Oncol202642431822PubTator, text miningPubMed
NSUN2-mediated m5C modification of E2F1 promotes lung adenocarcinoma progression via the RAD54L signaling axis.Respir Res202642399962PubTator, text miningPubMed
FBH1 and RAD54L directly interact and cooperate to drive replication fork reversal.bioRxiv202642239417PubTator, text miningPubMed
Ethnicity-specific molecular subtypes and a machine-learning risk model in Asian patients with non-muscle-invasive bladder cancer.Sci Rep202642215586PubTator, text miningPubMed
RAD54L promotes nascent DNA degradation and radial chromosome formation in FANC-deficient cells.bioRxiv202642182380PubTator, text miningPubMed

1 to 10 of 1,865

See all 1,865 papers

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this gene from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/genes/v1/8438

GET https://conductscience.org/api/genes/v1/8438/patents?page=2