Paper · 1379591

A naturally occurring secreted form of fibroblast growth factor (FGF) receptor 1 binds basic FGF in preference over acidic FGF.

Duan DS and 2 others. J Biol Chem, 1992. Cited by 72 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. A naturally occurring secreted form of fibroblast growth factor (FGF) receptor 1 binds basic FGF in preference over acidic FGF. Papers, record 1379591. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/1379591

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: A naturally occurring secreted form of fibroblast growth factor (FGF) receptor 1 binds basic FGF in preference over acidic FGF.; PubMed
Journal: J Biol Chem; PubMed
Year: 1992; PubMed
Publication types: Comparative Study, Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/1379591/; PubMed
The journal: https://conductscience.org/library/journal-index/2985121R; PubMed
Citations, by iCite: 72; iCite
Relative citation ratio, by iCite: 1.53; iCite
Revised in PubMed: 2021-02-10; PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Duan DS	D S
2	Werner S	S
3	Williams LT	L T

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Fibroblast Growth Factor 1	Major topic
Fibroblast Growth Factor 2	Major topic
Receptors, Cell Surface	Major topic
Animals
CHO Cells
Chickens
Cricetinae
Cross-Linking Reagents
Humans
Kinetics
Macromolecular Substances
Protein Conformation
Receptors, Fibroblast Growth Factor
Transfection

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
HL43821	NHLBI NIH HHS	United States	P01HL043821	PubMed and NIH

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by14

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 72 citations.

PMID	Title	Journal	Year	In Papers	Source
32,492,514	FGFR4: A promising therapeutic target for breast cancer and other solid tumors.	Pharmacol Ther	2020	https://conductscience.org/library/papers/32492514	PubMed
30,577,533	Targeting Cellular Trafficking of Fibroblast Growth Factor Receptors as a Strategy for Selective Cancer Treatment.	J Clin Med	2018	https://conductscience.org/library/papers/30577533	PubMed
25,772,309	The Fibroblast Growth Factor signaling pathway.	Wiley Interdiscip Rev Dev Biol	2015	https://conductscience.org/library/papers/25772309	PubMed
24,343,791	Fibroblast growth factor signaling in mammalian tooth development.	Odontology	2014	https://conductscience.org/library/papers/24343791	PubMed
21,830,216	Caveolin-1 orchestrates fibroblast growth factor 2 signaling control of angiogenesis in placental artery endothelial cell caveolae.	J Cell Physiol	2012	https://conductscience.org/library/papers/21830216	PubMed
20,889,570	Altered splicing of FGFR1 is associated with high tumor grade and stage and leads to increased sensitivity to FGF1 in bladder cancer.	Am J Pathol	2010	https://conductscience.org/library/papers/20889570	PubMed
18,216,218	Cellular signaling by fibroblast growth factors (FGFs) and their receptors (FGFRs) in male reproduction.	Endocr Rev	2008	https://conductscience.org/library/papers/18216218	PubMed
16,597,617	Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family.	J Biol Chem	2006	https://conductscience.org/library/papers/16597617	PubMed
9,763,444	Fibroblast growth factor (FGF) soluble receptor 1 acts as a natural inhibitor of FGF2 neurotrophic activity during retinal degeneration.	Mol Biol Cell	1998	https://conductscience.org/library/papers/9763444	PubMed
9,501,086	Soluble dominant-negative receptor uncovers essential roles for fibroblast growth factors in multi-organ induction and patterning.	EMBO J	1998	https://conductscience.org/library/papers/9501086	PubMed

1 to 10 of 14

See all 14 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
FGFR1	2,260	PubTator's text mining	https://conductscience.org/library/genes/2260

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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