Paper · 302186

Suppressive and enhancing T-cell factors as I-region gene products: properties and the subregion assignment.

Tada T and 2 others. Cold Spring Harb Symp Quant Biol, 1977. Cited by 62 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Suppressive and enhancing T-cell factors as I-region gene products: properties and the subregion assignment. Papers, record 302186. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/302186

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Suppressive and enhancing T-cell factors as I-region gene products: properties and the subregion assignment.; PubMed
Journal: Cold Spring Harb Symp Quant Biol; PubMed
Year: 1977; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/302186/; PubMed
DOI: https://doi.org/10.1101/sqb.1977.041.01.016; PubMed
The journal: https://conductscience.org/library/journal-index/1256107; PubMed
Citations, by iCite: 62; iCite
Relative citation ratio, by iCite: 1.46; iCite
Revised in PubMed: 2021-12-03; PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Tada T	T
2	Taniguchi M	M
3	David CS	C S

Source PubMed

Topics16

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Antibody Formation	Major topic
Genes	Major topic
Immunosuppression Therapy	Major topic
Isoantigens	Major topic
Lymphokines	Major topic
T-Lymphocytes	Major topic
Animals
Antilymphocyte Serum
Binding Sites
Cells, Cultured
Chromosome Mapping
Genetic Linkage
Histocompatibility Antigens
Immunologic Memory
Mice
Spleen

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by24

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 62 citations.

PMID	Title	Journal	Year	In Papers	Source
16,068,185	Structural polymorphism of I-E subregion antigens determined by a gene in the H-2K to I-B genetic interval.	Nature	1979	https://conductscience.org/library/papers/16068185	PubMed
7,020,898	Surface receptors on lymphoreticular cells: sensory devices for host recognition of foreign antigens and neoplasia.	Can Med Assoc J	1980	https://conductscience.org/library/papers/7020898	PubMed
6,972,996	Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self.	J Exp Med	1981	https://conductscience.org/library/papers/6972996	PubMed
6,966,320	Selective suppression of the major idiotypic component of an antihapten response by soluble T cell-derived factors with idiotypic or anti-idiotypic receptors.	J Exp Med	1980	https://conductscience.org/library/papers/6966320	PubMed
6,792,727	Helper and suppressor T cell factors.	Springer Semin Immunopathol	1980	https://conductscience.org/library/papers/6792727	PubMed
6,766,980	Cellular consequences in the suppression of antibody response by the antigen-specific T-cell factor.	J Exp Med	1980	https://conductscience.org/library/papers/6766980	PubMed
6,447,669	Antigen-binding receptors on T cells from long-term MLR. evidence of binding sites for allogeneic and self-MHC products.	Immunogenetics	1980	https://conductscience.org/library/papers/6447669	PubMed
6,445,400	Antigen-specific T cell-mediated suppression. V. H-2-linked genetic control of distinct antigen-specific defects in the production and activity of L-glutamic acid50-L-tyrosine50 suppressor factor.	J Exp Med	1980	https://conductscience.org/library/papers/6445400	PubMed
6,215,458	Mechanism responsible for the induction of I-J restriction on TS3 suppressor cells.	J Exp Med	1982	https://conductscience.org/library/papers/6215458	PubMed
6,189,952	IR gene regulation of the response to trinitrophenyl-polysaccharides. Two independent genes are required for antibody production.	J Exp Med	1983	https://conductscience.org/library/papers/6189952	PubMed

1 to 10 of 24

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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