Paper · 6972996

Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self.

Plate JM. J Exp Med, 1981. Cited by 9 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self. Papers, record 6972996. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6972996

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self.; PubMed
Journal: J Exp Med; PubMed
Year: 1981; PubMed
Publication types: Journal Article, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/6972996/; PubMed
DOI: https://doi.org/10.1084/jem.153.5.1102; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC2186169/; PubMed
The journal: https://conductscience.org/library/journal-index/2985109R; PubMed
Citations, by iCite: 9; iCite
Relative citation ratio, by iCite: 0.19; iCite
Revised in PubMed: 2019-05-08; PubMed

Authors1

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Plate JM	J M

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Major Histocompatibility Complex	Major topic
T-Lymphocytes	Major topic
Animals
Chromosome Mapping
Female
H-2 Antigens
Mice
Solubility

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
CA 25612	NCI NIH HHS	United States	R01CA025612	PubMed and NIH

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References23

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	4,587,740	A rapid method for the isolation of functional thymus-derived murine lymphocytes.	Eur J Immunol	1973	https://conductscience.org/library/papers/4587740	PubMed
2	4,547,153	Cell-mediated cytotoxicity to trinitrophenyl-modified syngeneic lymphocytes.	Eur J Immunol	1974	https://conductscience.org/library/papers/4547153	PubMed
3	123,260	The allogeneic effect: M-locus differences substitute for differences in the H-2 major histocompatibility complex.	J Immunol	1975	https://conductscience.org/library/papers/123260	PubMed
4	1,092,799	Functional subclasses of T lymphocytes bearing different Ly antigens. II. Cooperation between subclasses of Ly+ cells in the generation of killer activity.	J Exp Med	1975	https://conductscience.org/library/papers/1092799	PubMed
5	1,079,848	On the role of the H-2 histocompatibility complex in determining the specificity of cytotoxic effector cells sensitized against syngeneic trinitrophenyl-modified targets.	J Exp Med	1975	https://conductscience.org/library/papers/1079848	PubMed
6	52,685	Bifunctional major histocompatibility-linked genetic regulation of cell-mediated lympholysis to trinitrophenyl-modified autologous lymphocytes.	J Exp Med	1975	https://conductscience.org/library/papers/52685	PubMed
7	53,263	The major histocompatibility complex determines susceptibility to cytotoxic T cells directed against minor histocompatibility antigens.	J Exp Med	1975	https://conductscience.org/library/papers/53263	PubMed
8	58,463	Specificity of virus-immune effector T cells for H-2K or H-2D compatible interactions: implications for H-antigen diversity.	Transplant Rev	1976	https://conductscience.org/library/papers/58463	PubMed
9	1,086,882	Regulatory mechanisms in cell-mediated immune responses. IV. Expression of a receptor for mixed lymphocyte reaction suppressor factor on activated T lymphocytes.	J Exp Med	1976	https://conductscience.org/library/papers/1086882	PubMed
10	302,186	Suppressive and enhancing T-cell factors as I-region gene products: properties and the subregion assignment.	Cold Spring Harb Symp Quant Biol	1977	https://conductscience.org/library/papers/302186	PubMed

1 to 10 of 23

See all 23 references

Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 9 citations.

None in this release.

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
HLA-C	3,107	PubTator's text mining	https://conductscience.org/library/genes/3107

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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