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Paper324776

Regulation of the immune response. II. Repressor T cells in cyclophosphamide-induced tolerant mice.

Ramshaw IA and 2 others. Eur J Immunol, 1977. Cited by 69 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Regulation of the immune response. II. Repressor T cells in cyclophosphamide-induced tolerant mice. Papers, record 324776. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/324776

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Regulation of the immune response. II. Repressor T cells in cyclophosphamide-induced tolerant mice.
PubMed
Journal
Eur J Immunol
PubMed
Year
1977
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
69
iCite
Relative citation ratio, by iCite
1.59
iCite
Revised in PubMed
2021-12-03
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Ramshaw IAI A
2Bretscher PAP A
3Parish CRC R

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
CyclophosphamideMajor topic
Immune ToleranceMajor topic
Immunity, CellularMajor topic
T-LymphocytesMajor topic
Animals
Antilymphocyte Serum
Female
Hemolytic Plaque Technique
Horses
Hypersensitivity, Delayed
Immunosuppression Therapy
Lymphocytes
Mice
Mice, Inbred CBA

Source PubMed

Cited by15

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 69 citations.

PMIDTitleJournalYearIn PapersSource
31,231,378On Analyzing How the Th1/Th2 Phenotype of an Immune Response Is Determined: Classical Observations Must Not Be Ignored.Front Immunol2019https://conductscience.org/library/papers/31231378PubMed
24,684,592On the mechanism determining the TH1/TH2 phenotype of an immune response, and its pertinence to strategies for the prevention, and treatment, of certain infectious diseases.Scand J Immunol2014https://conductscience.org/library/papers/24684592PubMed
18,188,562Different immune correlates associated with tumor progression and regression: implications for prevention and treatment of cancer.Cancer Immunol Immunother2008https://conductscience.org/library/papers/18188562PubMed
15,819,695Increasing the foreignness of an antigen, by coupling a second and foreign antigen to it, increases the T helper type 2 component of the immune response to the first antigen.Immunology2005https://conductscience.org/library/papers/15819695PubMed
8,423,090Oral immunization of mice and swine with an attenuated Salmonella choleraesuis [delta cya-12 delta(crp-cdt)19] mutant containing a recombinant plasmid.Infect Immun1993https://conductscience.org/library/papers/8423090PubMed
6,976,414In vitro effects of 4-hydroperoxycyclophosphamide on human immunoregulatory T subset function. I. Selective effects on lymphocyte function in T-B cell collaboration.J Exp Med1982https://conductscience.org/library/papers/6976414PubMed
6,819,082The effects of cyclophosphamide on in vitro cytotoxic responses to a syngeneic tumour.Cancer Immunol Immunother1982https://conductscience.org/library/papers/6819082PubMed
3,849,980Chemoimmunotherapeutic effect of cyclophosphamide on the highly metastatic MAT 13762 tumor.Cancer Immunol Immunother1985https://conductscience.org/library/papers/3849980PubMed
3,724,838Experimental coccidioidomycosis: effects of cyclophosphamide in immunologic responses.Mycopathologia1986https://conductscience.org/library/papers/3724838PubMed
3,493,072A 6-thioguanine-resistant variant of the rat mammary adenocarcinoma 13762 that is more immunogenic.Cancer Immunol Immunother1987https://conductscience.org/library/papers/3493072PubMed
2,436,841Long-lasting enhancement of the delayed-type hypersensitivity response to heterologous erythrocytes in mice after a single injection of cyclophosphamide.Clin Exp Immunol1986https://conductscience.org/library/papers/2436841PubMed
1,828,396Inhibition of T suppressor cell function by local administration of an active cyclophosphamide derivative at the sensitization site.Clin Exp Immunol1991https://conductscience.org/library/papers/1828396PubMed
309,853Distinction between suppressors of the delayed-type hypersensitivity and the humoral response to sheep erythrocytes.Immunology1978https://conductscience.org/library/papers/309853PubMed
68,997Direct visualization of T lymphocytes bearing Ia antigens controlled by the I-J subregion.J Exp Med1977https://conductscience.org/library/papers/68997PubMed
33,118The capacity of microsomally-activated cyclophosphamide to induce immunosuppression in vitro.Immunology1978https://conductscience.org/library/papers/33118PubMed

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
  • Trials
  • Comments and corrections
  • References
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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