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Paper · 33223

The role of H-2 and Ia antigens in graft-versus-host reactions (GVHR). Presence of host alloantigens on donor cells after GVHR and suppression of GVHR with an anti-Ia antiserum against hose Ia antigens.

Prud'homme GJ and 2 others. J Exp Med, 1979. Cited by 45 papers, by iCite.

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ConductScience Library. The role of H-2 and Ia antigens in graft-versus-host reactions (GVHR). Presence of host alloantigens on donor cells after GVHR and suppression of GVHR with an anti-Ia antiserum against hose Ia antigens. Papers, record 33223. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/33223

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
The role of H-2 and Ia antigens in graft-versus-host reactions (GVHR). Presence of host alloantigens on donor cells after GVHR and suppression of GVHR with an anti-Ia antiserum against hose Ia antigens.
PubMed
Journal
J Exp Med
PubMed
Year
1979
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
45
iCite
Relative citation ratio, by iCite
1.24
iCite
Revised in PubMed
2021-12-03
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Prud'homme GJG J
2Sohn UU
3Delovitch TLT L

Source PubMed

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Graft vs Host ReactionMajor topic
H-2 AntigensMajor topic
IsoantigensMajor topic
Major Histocompatibility ComplexMajor topic
T-LymphocytesMajor topic
Animals
Antigens, Surface
Immunosuppression Therapy
Isoantibodies
Mice

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References31

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
11,085,338A new I subregion (I-J) marked by a locus (Ia-4) controlling surface determinants on suppressor T lymphocytes.J Exp Med1976https://conductscience.org/library/papers/1085338PubMed
21,092,798Functional subclasses of T-lymphocytes bearing different Ly antigens. I. The generation of functionally distinct T-cell subclasses is a differentiative process independent of antigen.J Exp Med1975https://conductscience.org/library/papers/1092798PubMed
31,092,799Functional subclasses of T lymphocytes bearing different Ly antigens. II. Cooperation between subclasses of Ly+ cells in the generation of killer activity.J Exp Med1975https://conductscience.org/library/papers/1092799PubMed
4129,499The role of nonclassical Fc receptor-associated, Ag-B antigens (Ia) in rat allograft enhancement.J Exp Med1976https://conductscience.org/library/papers/129499PubMed
5137,268Specific binding of K- and I-region products of the H-2 complex to activated thymus-derived (T) cells belonging to different Ly subclasses.J Exp Med1976https://conductscience.org/library/papers/137268PubMed
614,037,719Graft versus host reactions. Their natural history, and applicability as tools of research.Prog Allergy1962https://conductscience.org/library/papers/14037719PubMed
714,265,492SYNERGIC AND ANTAGONISTIC EFFECTS OF ISOANTIBODY UPON GRAFT-VERSUS-HOST DISEASE.Transplantation1965https://conductscience.org/library/papers/14265492PubMed
8142,791Activation of T and B lymphocytes in vitro. V. Cellular locus, metabolism and genetics of induction, and production of the allogeneic effect factor.J Immunol1977https://conductscience.org/library/papers/142791PubMed
917,471Prolongation of skin allograft survival and inhibition of graft versus host reaction in rodents treated with "middle molecules".Cell Immunol1977https://conductscience.org/library/papers/17471PubMed
10234,595Bone-marrow transplantation (first of two parts).N Engl J Med1975https://conductscience.org/library/papers/234595PubMed

1 to 10 of 31

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Cited by14

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 45 citations.

PMIDTitleJournalYearIn PapersSource
7,943,182Passive acquisition of leukocyte proteins is associated with changes in phosphorylation of cellular proteins and cell-cell adhesion properties.Am J Pathol1994https://conductscience.org/library/papers/7943182PubMed
7,320,686Protection against graft vs. host-associated immunosuppression in F1 mice. I. Activation of F1 regulatory cells by host-specific anti-major histocompatibility complex antibodies.J Exp Med1981https://conductscience.org/library/papers/7320686PubMed
7,033,532Induction and avoidance of graft-versus-host reactions: a review.J R Soc Med1981https://conductscience.org/library/papers/7033532PubMed
7,033,438Role of Ia antigens in graft vs. host reactions. II. Molecular and functional analysis of T cell alloreactivity by the characterization of host Ia antigens on alloactivated donor T cells.J Exp Med1982https://conductscience.org/library/papers/7033438PubMed
6,969,778
In vitro analysis of allogeneic lymphocyte interaction. V. Identification and characterization of two components of allogeneic effect factor, one of which displays H-2-restricted helper activity and the other, T cell-growth factor activity.
J Exp Med1981https://conductscience.org/library/papers/6969778PubMed
6,461,714
Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease.
J Exp Med1982https://conductscience.org/library/papers/6461714PubMed
6,403,650Graft-vs.-host-associated immune suppression is activated by recognition of allogeneic murine I-A antigens.J Exp Med1983https://conductscience.org/library/papers/6403650PubMed
6,340,519Cells bearing Ia antigens in the murine thymus. An ultrastructural study.Am J Pathol1983https://conductscience.org/library/papers/6340519PubMed
6,222,135Early development of the T cell repertoire. In vivo treatment of neonatal mice with anti-Ia antibodies interferes with differentiation of I-restricted T cells but not K/D-restricted T cells.J Exp Med1983https://conductscience.org/library/papers/6222135PubMed
6,212,625Conversion of immunity to suppression by in vivo administration of I-A subregion-specific antibodies.J Exp Med1982https://conductscience.org/library/papers/6212625PubMed

1 to 10 of 14

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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