ConductScience.org, home
Papers
API referenceFor AI assistants

Paper · 338932

"Host shutoff" function of bacteriophage T7: involvement of T7 gene 2 and gene 0.7 in the inactivation of Escherichia coli RNA polymerase.

Hesselbach BA and 1 other. J Virol, 1977. Cited by 63 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Download JSON
Cite this record

ConductScience Library. "Host shutoff" function of bacteriophage T7: involvement of T7 gene 2 and gene 0.7 in the inactivation of Escherichia coli RNA polymerase. Papers, record 338932. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/338932

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
"Host shutoff" function of bacteriophage T7: involvement of T7 gene 2 and gene 0.7 in the inactivation of Escherichia coli RNA polymerase.
PubMed
Journal
J Virol
PubMed
Year
1977
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
63
iCite
Relative citation ratio, by iCite
1.26
iCite
Revised in PubMed
2020-07-24
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Hesselbach BAB A
2Nakada DD

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
ColiphagesMajor topic
DNA-Directed RNA PolymerasesMajor topic
Escherichia coliMajor topic
Genes, ViralMajor topic
Viral ProteinsMajor topic
Kinetics
Mutation
Protein Kinases

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References20

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
1338,933I protein: bacteriophage T7-coded inhibitor of Escherichia coli RNA polymerase.J Virol1977https://conductscience.org/library/papers/338933PubMed
2797,534Bacteriophage T7 genetics.Curr Top Microbiol Immunol1976https://conductscience.org/library/papers/797534PubMed
34,614,077Control of gene expression in bacteriophage T7: transcriptional controls.Mol Gen Genet1974https://conductscience.org/library/papers/4614077PubMed
44,613,858Early to late switch in bacteriophage T7 development: functional decay of T7 early messenger RNA.J Mol Biol1974https://conductscience.org/library/papers/4613858PubMed
54,610,406Isolation of an inhibitor protein of E. coli RNA polymerase from T7 phage infected cell.Nature1974https://conductscience.org/library/papers/4610406PubMed
64,616,088The interaction bacterial and phage proteins with immobilized Escherichia coli RNA polymerase.J Mol Biol1974https://conductscience.org/library/papers/4616088PubMed
74,900,510A new method for the large scale purification of Escherichia coli deoxyribonucleic acid-dependent ribonucleic acid polymerase.J Biol Chem1969https://conductscience.org/library/papers/4900510PubMed
81,101,258In vivo and in vitro phosphorylation of DNA-dependent RNA polymerase of Escherichia coli by bacteriophage-T7-induced protein kinase.Proc Natl Acad Sci U S A1975https://conductscience.org/library/papers/1101258PubMed
91,104,891F-Factor-mediated restriction of bacteriophage T7: synthesis of RNA and protein in T7-infected Escherichia coli F- and F+ cells.J Virol1975https://conductscience.org/library/papers/1104891PubMed
101,105,195Inactive complex formation between E. coli RNA polymerase and inhibitor protein purified from T7 phage infected cells.Nature1975https://conductscience.org/library/papers/1105195PubMed

1 to 10 of 20

See all 20 references

Cited by27

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 63 citations.

PMIDTitleJournalYearIn PapersSource
42,889,960Metabolic Engineering and Bioprocess Optimization Strategies Promote l‑Azidohomoalanine Biosynthesis in Fed-Batch Bioreactor Culture.ACS Omega2026https://conductscience.org/library/papers/42889960PubMed
40,169,592A toxin-antitoxin system provides phage defense via DNA damage and repair.Nat Commun2025https://conductscience.org/library/papers/40169592PubMed
37,683,045Replication cycle timing determines phage sensitivity to a cytidine deaminase toxin/antitoxin bacterial defense system.PLoS Pathog2023https://conductscience.org/library/papers/37683045PubMed
33,838,104Shutoff of host transcription triggers a toxin-antitoxin system to cleave phage RNA and abort infection.Mol Cell2021https://conductscience.org/library/papers/33838104PubMed
32,887,931Identification of mycobacteriophage toxic genes reveals new features of mycobacterial physiology and morphology.Sci Rep2020https://conductscience.org/library/papers/32887931PubMed
30,766,528Predictable Molecular Adaptation of Coevolving Enterococcus faecium and Lytic Phage EfV12-phi1.Front Microbiol2018https://conductscience.org/library/papers/30766528PubMed
28,714,913Novel Fri1-like Viruses Infecting Acinetobacter baumannii-vB_AbaP_AS11 and vB_AbaP_AS12-Characterization, Comparative Genomic Analysis, and Host-Recognition Strategy.Viruses2017https://conductscience.org/library/papers/28714913PubMed
24,420,107Bacterial RNA polymerases: structural and functional relationships.World J Microbiol Biotechnol1993https://conductscience.org/library/papers/24420107PubMed
24,218,612Gene product 0.4 increases bacteriophage T7 competitiveness by inhibiting host cell division.Proc Natl Acad Sci U S A2013https://conductscience.org/library/papers/24218612PubMed
23,728,817Lytic infection of Lactococcus lactis by bacteriophages Tuc2009 and c2 triggers alternative transcriptional host responses.Appl Environ Microbiol2013https://conductscience.org/library/papers/23728817PubMed

1 to 10 of 27

See all 27 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/338932

GET https://conductscience.org/api/papers/v1/338932/cited_by?page=2

API referenceFor AI assistants