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Paper · 37511653

Molecular and Phenotypic Changes in FLExDUX4 Mice.

Murphy K and 3 others. J Pers Med, 2023. Cited by 6 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Molecular and Phenotypic Changes in FLExDUX4 Mice. Papers, record 37511653. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/37511653

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Molecular and Phenotypic Changes in FLExDUX4 Mice.
PubMed
Journal
J Pers Med
PubMed
Year
2023
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
6
iCite
Relative citation ratio, by iCite
0.73
iCite
Revised in PubMed
2025-07-19
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Murphy KKelly
2Zhang AAiping
3Bittel AJAdam Jhttps://orcid.org/0000-0003-2134-418X
4Chen YWYi-Wen

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated by
R21 HD103993NICHD NIH HHSUnited StatesR21HD103993PubMed

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References110

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
132,244,226Nrf2 deficiency promotes the increasing trend of autophagy during aging in skeletal muscle: a potential mechanism for the development of sarcopenia.Aging (Albany NY)2020https://conductscience.org/library/papers/32244226PubMed
215,674,778The D4Z4 repeat-mediated pathogenesis of facioscapulohumeral muscular dystrophy.Am J Hum Genet2005https://conductscience.org/library/papers/15674778PubMed
329,481,323NRF2 regulates core and stabilizing circadian clock loops, coupling redox and timekeeping in Mus musculus.Elife2018https://conductscience.org/library/papers/29481323PubMed
432,601,200Inhibition of DUX4 expression with antisense LNA gapmers as a therapy for facioscapulohumeral muscular dystrophy.Proc Natl Acad Sci U S A2020https://conductscience.org/library/papers/32601200PubMed
59,506,542Facioscapulohumeral dystrophy: a distinct regional myopathy with a novel molecular pathogenesis. FSH Consortium.Ann Neurol1998https://conductscience.org/library/papers/9506542PubMed
622,541,069A long ncRNA links copy number variation to a polycomb/trithorax epigenetic switch in FSHD muscular dystrophy.Cell2012https://conductscience.org/library/papers/22541069PubMed
725,031,281Differential DNA methylation of the D4Z4 repeat in patients with FSHD and asymptomatic carriers.Neurology2014https://conductscience.org/library/papers/25031281PubMed
826,951,377DUX4 recruits p300/CBP through its C-terminus and induces global H3K27 acetylation changes.Nucleic Acids Res2016https://conductscience.org/library/papers/26951377PubMed
922,796,148Functional muscle impairment in facioscapulohumeral muscular dystrophy is correlated with oxidative stress and mitochondrial dysfunction.Free Radic Biol Med2012https://conductscience.org/library/papers/22796148PubMed
1016,508,966Facioscapulohumeral muscular dystrophy.Muscle Nerve2006https://conductscience.org/library/papers/16508966PubMed

1 to 10 of 110

See all 110 references

Cited by4

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 6 citations.

PMIDTitleJournalYearIn PapersSource
41,536,811DUX4 reduction and muscle function improvement by subcutaneous delivery of gapmer antisense oligonucleotides.Mol Ther Nucleic Acids2026https://conductscience.org/library/papers/41536811PubMed
40,634,301Estrogen rescues muscle regeneration impaired by DUX4 in a humanized xenograft mouse model.Cell Death Dis2025https://conductscience.org/library/papers/40634301PubMed
39,518,930Molecular, Histological, and Functional Changes in Acta1-MCM;FLExDUX4/+ Mice.Int J Mol Sci2024https://conductscience.org/library/papers/39518930PubMed
39,000,102Systemic Pharmacotherapeutic Treatment of the ACTA1-MCM/FLExDUX4 Preclinical Mouse Model of FSHD.Int J Mol Sci2024https://conductscience.org/library/papers/39000102PubMed

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
DUX4100,288,687PubTator's text mininghttps://conductscience.org/library/genes/100288687

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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The record and each of its lists are JSON, twenty rows a page. No key.

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