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Paper · 39158595

New CRISPR/Cas9-based Fgfr2C361Y/+ mouse model of Crouzon syndrome exhibits skull and behavioral abnormalities.

Yue YY and 6 others. J Mol Med (Berl), 2024. Cited by 6 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. New CRISPR/Cas9-based Fgfr2C361Y/+ mouse model of Crouzon syndrome exhibits skull and behavioral abnormalities. Papers, record 39158595. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39158595

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
New CRISPR/Cas9-based Fgfr2C361Y/+ mouse model of Crouzon syndrome exhibits skull and behavioral abnormalities.
PubMed
Journal
J Mol Med (Berl)
PubMed
Year
2024
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
6
iCite
Relative citation ratio, by iCite
0.74, provisional
iCite
Revised in PubMed
2026-11-01
PubMed

Authors7

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Yue YYYing Yinghttps://orcid.org/0009-0000-8794-8803
2Lai CZChen-Zhi
3Guo XSXiao-Shuang
4Yang CSChang-Sheng
5Wang YYu
6Song GDGuo-Dong
7Jin XLXiao-Lei

Source PubMed

Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Receptor, Fibroblast Growth Factor, Type 2Major topic
Craniofacial DysostosisMajor topic
Disease Models, AnimalMajor topic
CRISPR-Cas SystemsMajor topic
SkullMajor topic
Animals
Mice
Phenotype
Behavior, Animal
Gene Editing
Male
Female

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References46

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
133,417,861Cranial Suture Regeneration Mitigates Skull and Neurocognitive Defects in Craniosynostosis.Cell2021https://conductscience.org/library/papers/33417861PubMed
229,236,889Syndromic craniosynostosis: neuropsycholinguistic abilities and imaging analysis of the central nervous system.Arq Neuropsiquiatr2017https://conductscience.org/library/papers/29236889PubMed
321,248,745Craniosynostosis.Eur J Hum Genet2011https://conductscience.org/library/papers/21248745PubMed
438,369,396Association of cranial base suture/synchondrosis fusion with severity of increased intracranial pressure in Crouzon syndrome.J Craniomaxillofac Surg2024https://conductscience.org/library/papers/38369396PubMed
520,643,727Prevalence and complications of single-gene and chromosomal disorders in craniosynostosis.Pediatrics2010https://conductscience.org/library/papers/20643727PubMed
614,499,350A Ser252Trp [corrected] substitution in mouse fibroblast growth factor receptor 2 (Fgfr2) results in craniosynostosis.Bone2003https://conductscience.org/library/papers/14499350PubMed
720,503,384The missense mutation W290R in Fgfr2 causes developmental defects from aberrant IIIb and IIIc signaling.Dev Dyn2010https://conductscience.org/library/papers/20503384PubMed
820,077,479Brain phenotypes in two FGFR2 mouse models for Apert syndrome.Dev Dyn2010https://conductscience.org/library/papers/20077479PubMed
935,254,402FGFR3 overactivation in the brain is responsible for memory impairments in Crouzon syndrome mouse model.J Exp Med2022https://conductscience.org/library/papers/35254402PubMed
1030,633,021Analysis of Airway and Midface in Crouzon Syndromes.Ann Plast Surg2019https://conductscience.org/library/papers/30633021PubMed

1 to 10 of 46

See all 46 references

Cited by6

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 6 citations.

PMIDTitleJournalYearIn PapersSource
42,615,907Fzd7 Restrains Pink1-Dependent Mitophagy in Suture Stem Cells to Maintain Cranial Suture Patency.Adv Sci (Weinh)2026https://conductscience.org/library/papers/42615907PubMed
42,258,016Mitochondria-related gene and protein changes in craniosynostosis: integrated transcriptomics and Fgfr2C361Y/+ cranial suture proteomics.Funct Integr Genomics2026https://conductscience.org/library/papers/42258016PubMed
42,071,254Spatiotemporal single-cell atlas of suture stem cell dynamics in craniosynostosis.Stem Cell Res Ther2026https://conductscience.org/library/papers/42071254PubMed
41,768,990Human cranial stem cells: developmental, pathologic, and therapeutic implications.Front Cell Dev Biol2026https://conductscience.org/library/papers/41768990PubMed
41,584,352Unraveling the FGFR-RNA splicing axis: Mechanisms, oncogenic crosstalks and innovations for therapeutic purpose.Acta Pharm Sin B2026https://conductscience.org/library/papers/41584352PubMed
41,390,905P38α MAPK-induced senescence in cranial suture progenitor cells promotes craniosynostosis.Commun Biol2025https://conductscience.org/library/papers/41390905PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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