Paper · 39216481
A conserved transcription factor regulatory program promotes tendon fate.
Niu X and 13 others. Dev Cell, 2024. Cited by 6 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
Cite this record
ConductScience Library. A conserved transcription factor regulatory program promotes tendon fate. Papers, record 39216481. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39216481
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Publication types
- Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural
- PubMed
Topics14
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Zebrafish | Major topic |
| Tendons | Major topic |
| Zebrafish Proteins | Major topic |
| Transcription Factors | Major topic |
| Cyclic AMP Response Element-Binding Protein | Major topic |
| Cell Differentiation | Major topic |
| Animals | |
| Humans | |
| Mice | |
| Gene Expression Regulation, Developmental | |
| Colforsin | |
| Cyclic AMP | |
| Pluripotent Stem Cells | |
| Enhancer Elements, Genetic |
Source PubMed
Grants5
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by | Source |
|---|---|---|---|---|---|
| R03 DE024771 | NIDCR NIH HHS | United States | R03DE024771 | PubMed | NIH RePORTER |
| R00 HD069533 | NICHD NIH HHS | United States | R00HD069533 | PubMed | NIH RePORTER |
| R01 AR074541 | NIAMS NIH HHS | United States | R01AR074541 | PubMed | NIH RePORTER |
| R01 AR079495 | NIAMS NIH HHS | United States | R01AR079495 | PubMed | NIH RePORTER |
| R01 AR073821 | NIAMS NIH HHS | United States | R01AR073821 | PubMed | NIH RePORTER |
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References89
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 32,463,804 | Scleraxis is required for the growth of adult tendons in response to mechanical loading. | JCI Insight | 2020 | https://conductscience.org/library/papers/32463804 | PubMed |
| 2 | 20,056,790 | High-throughput cell transplantation establishes that tumor-initiating cells are abundant in zebrafish T-cell acute lymphoblastic leukemia. | Blood | 2010 | https://conductscience.org/library/papers/20056790 | PubMed |
| 3 | 33,712,729 | Creb5 establishes the competence for Prg4 expression in articular cartilage. | Commun Biol | 2021 | https://conductscience.org/library/papers/33712729 | PubMed |
| 4 | 24,076,664 | Activation of Hedgehog signaling by loss of GNAS causes heterotopic ossification. | Nat Med | 2013 | https://conductscience.org/library/papers/24076664 | PubMed |
| 5 | 36,905,926 | Nuclear receptor Nr5a2 promotes diverse connective tissue fates in the jaw. | Dev Cell | 2023 | https://conductscience.org/library/papers/36905926 | PubMed |
| 6 | 29,644,940 | Transient Scleraxis Overexpression Combined with Cyclic Strain Enhances Ligament Cell Differentiation. | Tissue Eng Part A | 2018 | https://conductscience.org/library/papers/29644940 | PubMed |
| 7 | 22,491,032 | Enhancers: emerging roles in cell fate specification. | EMBO Rep | 2012 | https://conductscience.org/library/papers/22491032 | PubMed |
| 8 | 26,062,940 | Musculoskeletal integration at the wrist underlies the modular development of limb tendons. | Development | 2015 | https://conductscience.org/library/papers/26062940 | PubMed |
| 9 | 23,095,209 | Neuromuscular symptoms in a patient with familial pseudohypoparathyroidism type Ib diagnosed by methylation-specific multiplex ligation-dependent probe amplification. | Endocr J | 2013 | https://conductscience.org/library/papers/23095209 | PubMed |
| 10 | 12,086,472 | Fgf4 positively regulates scleraxis and tenascin expression in chick limb tendons. | Dev Biol | 2002 | https://conductscience.org/library/papers/12086472 | PubMed |
1 to 10 of 89
See all 89 referencesCited by4
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 6 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 41,755,020 | Psoralen Promotes Direct Chemical Reprogramming of Mouse Embryonic Fibroblasts into Osteoblast-like Cells. | Pharmaceutics | 2026 | https://conductscience.org/library/papers/41755020 | PubMed |
| 41,607,347 | Gene regulatory programs underlying diversification of facial ligaments and tendons in zebrafish. | Development | 2026 | https://conductscience.org/library/papers/41607347 | PubMed |
| 41,582,829 | Analysis of cranial tenocyte heterogeneity reveals a role for Wnt signaling in tendon attachments. | Development | 2026 | https://conductscience.org/library/papers/41582829 | PubMed |
| 41,135,529 | The roles of TGF-β, Wnt, and MAPK signaling pathways in joint lineage specification in vitro and ex vivo. | Stem Cell Reports | 2025 | https://conductscience.org/library/papers/41135529 | PubMed |
Genes1
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes |
|---|---|---|---|
| EBF1 | 1,879 | PubTator's text mining | https://conductscience.org/library/genes/1879 |
Source NCBI Gene
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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