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Paper · 39376558

Regulating the lncRNA DSCR9/RPLP2/PI3K/AKT axis: an important mechanism of Xinfeng capsules in improving rheumatoid arthritis.

Wang F and 1 other. Front Immunol, 2024. Cited by 16 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Regulating the lncRNA DSCR9/RPLP2/PI3K/AKT axis: an important mechanism of Xinfeng capsules in improving rheumatoid arthritis. Papers, record 39376558. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39376558

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Regulating the lncRNA DSCR9/RPLP2/PI3K/AKT axis: an important mechanism of Xinfeng capsules in improving rheumatoid arthritis.
PubMed
Journal
Front Immunol
PubMed
Year
2024
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
16
iCite
Relative citation ratio, by iCite
3.56, provisional
iCite
Revised in PubMed
2026-10-30
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Wang FFanfan
2Liu JJian

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Arthritis, RheumatoidMajor topic
RNA, Long NoncodingMajor topic
Proto-Oncogene Proteins c-aktMajor topic
Phosphatidylinositol 3-KinasesMajor topic
Drugs, Chinese HerbalMajor topic
Signal TransductionMajor topic
Humans
Animals
Male
Female
Rats
Middle Aged
Arthritis, Experimental
Capsules

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References65

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
19,082,942Adjuvant arthritis as a model of inflammatory cachexia.Arthritis Rheum1997https://conductscience.org/library/papers/9082942PubMed
232,253,838Use of Tripterygium wilfordii Hook F for immune-mediated inflammatory diseases: progress and future prospects.J Zhejiang Univ Sci B2020https://conductscience.org/library/papers/32253838PubMed
328,515,692Wu-Tou Decoction in Rheumatoid Arthritis: Integrating Network Pharmacology and In Vivo Pharmacological Evaluation.Front Pharmacol2017https://conductscience.org/library/papers/28515692PubMed
429,351,810Identify Down syndrome transcriptome associations using integrative analysis of microarray database and correlation-interaction network.Hum Genomics2018https://conductscience.org/library/papers/29351810PubMed
529,417,936Rheumatoid arthritis.Nat Rev Dis Primers2018https://conductscience.org/library/papers/29417936PubMed
626,547,329[Effect of triptolide on expressions of Notch receptors and ligands in rats with adjuvant- induced arthritis and reduced pulmonary function].Nan Fang Yi Ke Da Xue Xue Bao2015https://conductscience.org/library/papers/26547329PubMed
733,333,049Nimbolide exerts protective effects in complete Freund's adjuvant induced inflammatory arthritis via abrogation of STAT-3/NF-κB/Notch-1 signaling.Life Sci2021https://conductscience.org/library/papers/33333049PubMed
820,193,003Fibroblast-like synoviocytes: key effector cells in rheumatoid arthritis.Immunol Rev2010https://conductscience.org/library/papers/20193003PubMed
930,767,875Neutrophil-derived lactoferrin induces the inflammatory responses of rheumatoid arthritis synovial fibroblasts via Toll-like receptor 4.Clin Exp Rheumatol2019https://conductscience.org/library/papers/30767875PubMed
1024,982,240Endogenous MMP-9 and not MMP-2 promotes rheumatoid synovial fibroblast survival, inflammation and cartilage degradation.Rheumatology (Oxford)2014https://conductscience.org/library/papers/24982240PubMed

1 to 10 of 65

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Cited by13

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 16 citations.

PMIDTitleJournalYearIn PapersSource
42,610,193Therapeutic Properties and Underlying Mechanisms of Traditional Chinese Medicine in Managing Rheumatoid Arthritis: An Integrative Review of Clinical and Molecular Findings.Phytother Res2026https://conductscience.org/library/papers/42610193PubMed
42,169,647Role of the PI3K/AKT signaling pathway in regulating gene expression in rheumatoid arthritis (Review).Mol Med Rep2026https://conductscience.org/library/papers/42169647PubMed
41,960,192Frontier research on mechanisms of bone destruction in rheumatoid arthritis.Front Cell Dev Biol2026https://conductscience.org/library/papers/41960192PubMed
41,695,173Multidimensional effects of the Xin'an Jianpi Tongbi Formula on self-perception of patients with rheumatoid arthritis: focusing on the mediating role of systemic inflammation index.Front Med (Lausanne)2026https://conductscience.org/library/papers/41695173PubMed
41,585,231Baricitinib in chronic kidney disease: an exploratory analysis integrating network toxicology, molecular docking and pharmacovigilance.Front Med (Lausanne)2025https://conductscience.org/library/papers/41585231PubMed
41,488,659
Joint association between pan-immune inflammation value and physical pain with self-perception of patients in rheumatoid arthritis: a retrospective cohort study employing traditional statistics and interpretable machine learning.
Front Immunol2025https://conductscience.org/library/papers/41488659PubMed
41,414,679Fedratinib in chronic kidney disease: antifibrotic potential and renal safety signals from integrated network toxicology and pharmacovigilance.Ren Fail2025https://conductscience.org/library/papers/41414679PubMed
40,771,812Mechanistic and therapeutic insights into the function of different cell death modalities in rheumatoid arthritis: emphasis on the crosstalk with non-coding RNAs.Front Immunol2025https://conductscience.org/library/papers/40771812PubMed
40,453,089Novel insights into the crosstalk between non-coding RNA and apoptosis in rheumatoid arthritis: diagnostic functions and therapeutic applications.Front Immunol2025https://conductscience.org/library/papers/40453089PubMed
40,453,080New insights into the role of cellular senescence and rheumatic diseases.Front Immunol2025https://conductscience.org/library/papers/40453080PubMed

1 to 10 of 13

See all 13 cited by

Genes3

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
AKT1207PubTator's text mininghttps://conductscience.org/library/genes/207NCBI Gene
DSCR9257,203PubTator's text mininghttps://conductscience.org/library/genes/257203NCBI Gene
RPLP26,181PubTator's text mininghttps://conductscience.org/library/genes/6181NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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