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Paper · 41008530

Beneficial Effects of Different Types of Exercise on Diabetic Cardiomyopathy.

Ma X and 8 others. Biomolecules, 2025. Cited by 7 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Beneficial Effects of Different Types of Exercise on Diabetic Cardiomyopathy. Papers, record 41008530. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41008530

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Beneficial Effects of Different Types of Exercise on Diabetic Cardiomyopathy.
PubMed
Journal
Biomolecules
PubMed
Year
2025
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
7
iCite
Relative citation ratio, by iCite
2.0, provisional
iCite
Revised in PubMed
2026-03-29
PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Ma XXiaotong
2Gao HHaoyang
3Wang ZZe
4Zhu DDanlin
5Dai WWei
6Wu MMingyu
7Guo YYifan
8Zhao LLinlin
9Xiao WWeihua

Source PubMed

Topics16

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Diabetic CardiomyopathiesMajor topic
Physical Conditioning, AnimalMajor topic
Animals
Male
Mice
Mice, Inbred C57BL
Diabetes Mellitus, Experimental
Myocardium
Signal Transduction
Myocytes, Cardiac
Fibrosis
Diabetes Mellitus, Type 2
Diet, High-Fat
Cytokines
Resistance Training
High-Intensity Interval Training

Source PubMed

Grants6

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
32371185the National Natural Science Foundation of ChinaPubMedPubMed
Nothe Shanghai Oriental Talents Program (Youth Project)PubMedPubMed
23010504200the Shanghai Science and Technology Plan ProjectPubMedPubMed
2025KF002the Key Lab of Exercise and Health Sciences of Ministry of Education (Shanghai University of Sport)PubMedPubMed
NO. 11DZ2261100the Shanghai Key Lab of Human Performance (Shanghai University of Sport)PubMedPubMed
YJSCX-2025-012the Research and Innovation Grant for Graduate Students (Shanghai University of Sport)YJSCX202501PubMedNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References66

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
135,909,160Cardiomyocyte-specific knockout of ADAM17 ameliorates left ventricular remodeling and function in diabetic cardiomyopathy of mice.Signal Transduct Target Ther2022https://conductscience.org/library/papers/35909160PubMed
236,568,097Diabetic cardiomyopathy: Clinical phenotype and practice.Front Endocrinol (Lausanne)2022https://conductscience.org/library/papers/36568097PubMed
339,250,437Swimming alleviates myocardial fibrosis of type II diabetic rats through activating miR-34a-mediated SIRT1/PGC-1α/FNDC5 signal pathway.PLoS One2024https://conductscience.org/library/papers/39250437PubMed
438,329,809TGF-β as a therapeutic target in the infarcted and failing heart: cellular mechanisms, challenges, and opportunities.Expert Opin Ther Targets2024https://conductscience.org/library/papers/38329809PubMed
532,922,365High-Intensity Interval Training Restores Glycolipid Metabolism and Mitochondrial Function in Skeletal Muscle of Mice With Type 2 Diabetes.Front Endocrinol (Lausanne)2020https://conductscience.org/library/papers/32922365PubMed
638,698,281Inhibition of fatty acid uptake by TGR5 prevents diabetic cardiomyopathy.Nat Metab2024https://conductscience.org/library/papers/38698281PubMed
736,675,183ATF3/SPI1/SLC31A1 Signaling Promotes Cuproptosis Induced by Advanced Glycosylation End Products in Diabetic Myocardial Injury.Int J Mol Sci2023https://conductscience.org/library/papers/36675183PubMed
829,032,504Resistance exercise improves cardiac function and mitochondrial efficiency in diabetic rat hearts.Pflugers Arch2018https://conductscience.org/library/papers/29032504PubMed
925,136,835NLRP3 gene silencing ameliorates diabetic cardiomyopathy in a type 2 diabetes rat model.PLoS One2014https://conductscience.org/library/papers/25136835PubMed
1035,235,096Lipotoxicity-induced mtDNA release promotes diabetic cardiomyopathy by activating the cGAS-STING pathway in obesity-related diabetes.Cell Biol Toxicol2023https://conductscience.org/library/papers/35235096PubMed

1 to 10 of 66

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Cited by11

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 7 citations.

PMIDTitleJournalYearIn PapersSource
42,963,201Exercise-Based Management of Diabetic Cardiomyopathy: Preclinical Mechanisms, Clinical Evidence, and Translational Strategies.J Diabetes2026https://conductscience.org/library/papers/42963201PubMed
42,955,468Combined crocin and treadmill exercise regulate cardiac MicroRNA-23b, nuclear factor Erythroid 2-related factor 2, and tumor necrosis factor-alpha in experimental type 2 diabetes mellitus.Mol Biol Rep2026https://conductscience.org/library/papers/42955468PubMed
42,942,053Exercise-Mediated Regulation of Ferroptosis in Diabetic Cardiomyopathy: Mechanisms and Therapeutic Perspectives.Int J Mol Sci2026https://conductscience.org/library/papers/42942053PubMed
42,934,109[Aerobic exercise improves metabolic disorders and multi-organ damage in diabetic rats by activating the Sirt1/Creb1/Ppargc1a axis].Nan Fang Yi Ke Da Xue Xue Bao2026https://conductscience.org/library/papers/42934109PubMed
42,757,104Role of the cGAS-STING signaling pathway in diabetes mellitus and its complications: from mechanisms to therapeutics.Front Pharmacol2026https://conductscience.org/library/papers/42757104PubMed
42,198,977[Melatonin alleviates cardiomyocyte necroptosis in diabetic mice by inhibiting the STING signaling pathway].Nan Fang Yi Ke Da Xue Xue Bao2026https://conductscience.org/library/papers/42198977PubMed
41,832,545Exercise modulates redox homeostasis in cardiovascular and metabolic diseases: from bench to clinic.Cardiovasc Diabetol2026https://conductscience.org/library/papers/41832545PubMed
41,704,821Mitochondria: the central hub linking exercise to enhanced cardiac function.Front Physiol2026https://conductscience.org/library/papers/41704821PubMed
41,695,095Aerobic exercise inhibits oxidative stress and improves diabetic cardiomyopathy in rats by activating the PROC/PAR1/Nrf2/HO-1 signaling pathway.Front Physiol2026https://conductscience.org/library/papers/41695095PubMed
41,594,656BDH1 Mediates Aerobic Exercise-Induced Improvement in Skeletal Muscle Metabolic Remodeling in Type 2 Diabetes Mellitus.Biomolecules2026https://conductscience.org/library/papers/41594656PubMed

1 to 10 of 11

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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